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Tafamidis (Fx-1006A): a first-in-class disease-modifying therapy for transthyretin familial amyloid
被引:0
|作者:
Coelho, T.
[1
]
Maia, L.
[1
]
Martins da Silva, A.
[1
]
Waddingtion-Cruz, M.
[2
]
Plante-Bordeneuve, V.
[3
]
Lozeron, P.
[4
]
Suhr, O. B.
[5
]
Campistol, J.
[6
]
Conceicao, I. M.
[7
]
Schmidt, H.
[8
]
Trigo, P.
[9
]
Packman, J.
[10
]
Grogan, D. R.
[10
]
机构:
[1] Hosp Santo Antonio, Dept Neurophysiol, Oporto, Portugal
[2] Univ Fed Rio de Janeiro, Univ Hosp, Neurol Serv, BR-21941 Rio De Janeiro, Brazil
[3] CHU Henri Mondor, Dept Neurol, F-94010 Creteil, France
[4] CHU Bicetre, Dept Neurol, Le Kremlin Bicetre, France
[5] Umea Univ Hosp, Dept Publ Hlth & Clin Med, S-90185 Umea, Sweden
[6] Hosp Clin Barcelona, Renal Transplant Unit, Barcelona, Spain
[7] Hosp Santa Maria, Dept Neurol, Lisbon, Portugal
[8] Univ Klinikum Munster, Munster, Germany
[9] FLENI, Buenos Aires, DF, Argentina
[10] FoldRx Pharmaceut Inc, Cambridge, MA USA
来源:
AMYLOID-JOURNAL OF PROTEIN FOLDING DISORDERS
|
2010年
/
17卷
关键词:
D O I:
暂无
中图分类号:
Q5 [生物化学];
Q7 [分子生物学];
学科分类号:
071010 ;
081704 ;
摘要:
引用
收藏
页码:75 / 76
页数:2
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