Stat5 Is Essential for BCR-ABL-Transformed Chronic Myeloid Leukemia (CML) Associated with Increased CCN3 Gene Expression.

被引：0

作者：

Zhu, Bing-Mei ^{[1
]}

Wickre, Mark ^{[1
]}

Na, Risu ^{[1
]}

Klover, Peter ^{[1
]}

Martin, Cyril ^{[1
]}

Kimura, Akiko ^{[1
]}

Hennighausen, Lothar ^{[1
]}

机构：

[1] NIDDK, Lab Genet & Physiol, NIH, Bethesda, MD USA

来源：

BLOOD | 2009年 / 114卷 / 22期

关键词：

D O I：

暂无

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：1266 / 1266

页数：1

共 29 条

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[22] EXPRESSION OF THE CHRONIC MYELOGENOUS LEUKEMIA-ASSOCIATED P210BCR/ABL ONCOPROTEIN IN A MURINE IL-3 DEPENDENT MYELOID CELL-LINE
LANEUVILLE, P
HEISTERKAMP, N
GROFFEN, J
ONCOGENE, 1991, 6 (02) : 275 - 282
[23] Pioglitazone Did Not Affect PPAR-Γ, STAT5, HIF2α and CITED2 Gene Expression in Chronic Myeloid Leukemia Patients with Deep Molecular Response
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[24] MAPPING OF BREAKPOINTS, AND RELATIONSHIP TO BCR-ABL RNA EXPRESSION, IN PHILADELPHIA-CHROMOSOME-POSITIVE CHRONIC MYELOID-LEUKEMIA PATIENTS WITH A BREAKPOINT AROUND EXON-14 (B3) OF THE BCR GENE
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SPROUL, AM
LEIBOWITZ, D
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LEUKEMIA, 1991, 5 (11) : 937 - 941
[25] Polyphenol tri-vanillic ester 13c inhibits P-JAK2V617F and Bcr-Abl oncokinase expression in correlation with STAT3/STAT5 inactivation and apoptosis induction in human leukemia cells
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[26] Dasatinib and Imatinib-Induced Reductions in BCR-ABL Transcript Levels Below 10% At 3 Months Are Associated with Improved Responses in Patients with Newly Diagnosed Chronic Myeloid Leukemia in Chronic Phase (CML-CP): Analysis of Molecular Response Kinetics in the DASISION Trial
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[27] Increased Frequency of Circulating Activated FOXP3+ Regulatory T Cell Subset in Patients with Chronic Lymphocytic Leukemia Is Associated with the Estimate of the Size of the Tumor Mass, STAT5 Signaling and Disease Course during Follow-Up of Patients on Therapy
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[28] Interferon Alpha 2a (IFN) Maintenance Therapy After Imatinib Plus IFN Induction Therapy in Chronic Myeloid Leukemia (CML) Induces Stable Long-Term Molecular Remissions and Is Associated with Increased Proteinase 3 (PR3) Expression and the Presence of PR1-Specific T-Cells
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[29] Downregulation of Mir-146a-5p, Mir-99b-5p, Mir-143-3p, Mir-10a-5p and Mir-151a-3p Associated with PI3K/AKT, p53, NF-Kb, and Fanconi Anemia/BRCA Signaling Pathways Are Observed in Imatinib-Resistant Chronic Myeloid Leukemia Patients without Detectable BCR-ABL kinase Domain Mutations
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