The proapoptotic BCL-2 family member BIM mediates motoneuron loss in a model of amyotrophic lateral sclerosis

被引：64
|
作者：
Hetz, C.
Thielen, P.
Fisher, J.
Pasinelli, P.
Brown, R. H.
Korsmeyer, S.
Glimcher, L.
机构：
[1] Harvard Univ, Sch Publ Hlth, Dept Immunol & Infect Dis, Boston, MA 02115 USA
[2] Univ Chile, Dept Cellular & Mol Biol, Inst Biomed Sci, FONDAP Ctr Mol Studies Cell, Santiago, Chile
[3] Harvard Univ, Sch Med, Dana Farber Canc Inst, Dept Canc Immunol & AIDS,Howard Hughes Med Inst, Boston, MA 02115 USA
[4] Thomas Jefferson Univ, Frances & Joseph Weinberg Unit ALS Res, Farber Inst Neurosci, Philadelphia, PA 19107 USA
[5] Harvard Univ, Sch Med, Dept Med, Boston, MA 02115 USA
来源：
CELL DEATH AND DIFFERENTIATION | 2007年 / 14卷 / 07期
关键词：
D O I：
10.1038/sj.cdd.4402166
中图分类号：
Q5 [生物化学]; Q7 [分子生物学];
学科分类号：
071010 ; 081704 ;
摘要：
引用
收藏
页码：1386 / 1389
页数：4
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