Behavioral and Molecular Effects of Endogenic Kynurenines Deficit in the Honeybee (Apis mellifera L.)

被引：0

作者：

Lopatina, N. G. ^{[1
]}

Zachepilo, T. G. ^{[1
]}

Chesnokova, E. G. ^{[1
]}

Savvateeva-Popova, E. V. ^{[1
]}

机构：

[1] Russian Acad Sci, IP Pavlov Physiol Inst, St Petersburg, Russia

来源：

ZHURNAL VYSSHEI NERVNOI DEYATELNOSTI IMENI I P PAVLOVA | 2010年 / 60卷 / 02期

关键词：

kynurenines; long-term memory; LIMK1; F-actin; honeybee; AMINOTRANSFERASE-II; HUNTINGTONS-DISEASE; ACTIN CYTOSKELETON; MORPHOLOGY; RECEPTORS; BRAIN; MICE; LTP;

D O I：

暂无

中图分类号：

Q189 [神经科学];

学科分类号：

071006 ;

摘要：

The importance of tryptophan endogenous metabolites kynurenines in the long-term memory and functioning of the signal cascade GluR - LIMK1 - F-actin, mediating the long-term memory trace storage, was demonstrated. The deficit of kynurenines induced by allopurinol (tryptophanoxygenase inhibitor) suppressed the long-term memory, decreased the LIMK1 expression, and paradoxically increased the F-actin content in the honeybee brain. These data agree with the earlier findings in drosophila mutant vermilion (the mutation of tryptophanoxygenase gene).

引用

页码：229 / 235

页数：7

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