Coronary Artery Ectasia: Review of the Non-Atherosclerotic Molecular and Pathophysiologic Concepts

被引：12
|
作者：
Richards, Gavin H. C. [1 ]
Hong, Kathryn L. [1 ,2 ]
Henein, Michael Y. [3 ]
Hanratty, Colm [1 ]
Boles, Usama [1 ,4 ]
机构：
[1] Mater Private Hosp, Cardiovasc Res Inst CVRI Dublin, Dublin D07 WKW8, Ireland
[2] Univ Coll Dublin, Sch Med, Dublin D04 V1W8, Ireland
[3] Umea Univ, Dept Publ Hlth & Clin Med, Heart Clin, S-90187 Umea, Sweden
[4] Tipperary Univ Hosp, Dept Cardiol, Clonmel E91 VY40, Ireland
来源：
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES | 2022年 / 23卷 / 09期
关键词：
coronary artery ectasia; coronary artery aneurysm; CAD; cytokine; lipidome; RISK; METABOLISM; CYTOKINES; DIAGNOSIS;
D O I：
10.3390/ijms23095195
中图分类号：
Q5 [生物化学]; Q7 [分子生物学];
学科分类号：
071010 ; 081704 ;
摘要：
Coronary artery ectasia (CAE) is frequently encountered in clinical practice, conjointly with atherosclerotic CAD (CAD). Given the overlapping cardiovascular risk factors for patients with concomitant CAE and atherosclerotic CAD, a common underlying pathophysiology is often postulated. However, coronary artery ectasia may arise independently, as isolated (pure) CAE, thereby raising suspicions of an alternative mechanism. Herein, we review the existing evidence for the pathophysiology of CAE in order to help direct management strategies towards enhanced detection and treatment.
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页数：11
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