Ca2+ transport across the tonoplast of intact vacuoles from Chenopodium album L suspension cells: ATP-dependent import and IP3 induced release.

被引:0
|
作者
Lommel, C [1 ]
Felle, HH [1 ]
机构
[1] UNIV GIESSEN,D-35390 GIESSEN,GERMANY
关键词
D O I
暂无
中图分类号
Q94 [植物学];
学科分类号
071001 ;
摘要
引用
收藏
页码:1435 / 1435
页数:1
相关论文
共 34 条
  • [21] ATP-induced beta-glucuronidase release from undifferentiated HL-60 cells is dependent on Ca2+ ions
    Packham, DE
    Jiang, LL
    Conigrave, AD
    CELLULAR SIGNALLING, 1996, 8 (01) : 67 - 73
  • [22] Not type-3, but type-1 inositol trisphosphate (IP3) receptors is crucial for IP3-Induced Ca2+ release (IICR) and proliferation in vascular smooth muscle cells (VSMC)
    Wang, Y
    Chen, J
    Wang, Y
    Toshiaki, N
    Kuniaki, I
    Shin, WS
    Sakaki, Y
    Toyo-oka, T
    CIRCULATION, 1999, 100 (18) : 209 - 209
  • [23] BRADYKININ (BK)-INDUCED CA2+ INFLUX INHIBITS CA2+ RELEASE FROM THE INOSITOL-1,4,5-TRISPHOSPHATE(IP3)-SENSITIVE INTRACELLULAR STORE IN CALF PULMONARY-ARTERY ENDOTHELIAL-CELLS
    SCHILLING, WP
    CABELLO, O
    FASEB JOURNAL, 1993, 7 (03): : A131 - A131
  • [24] Down-regulation of type-1 inositol trisphosphate (IP3) receptors (IP3R1) and functional IP3-induced Ca2+ release (IICR) in vascular smooth muscle cells (VSMC) by basal release of nitric oxide (NO) from endothelial cells (EC)
    Wang, Y
    Wang, Y
    Chen, J
    Nakajima, T
    Iwasawa, K
    Shin, WS
    Sakaki, Y
    Toyo-Oka, T
    CIRCULATION, 1999, 100 (18) : 623 - 623
  • [25] Crucial role of type 1, but not type 3, inositol 1,4,5-trisphosphate (IP3) receptors in IP3-induced Ca2+ release, capacitative Ca2+ entry, and proliferation of A7r5 vascular smooth muscle cells
    Wang, YP
    Chen, J
    Wang, Y
    Taylor, CW
    Hirata, Y
    Hagiwara, H
    Mikoshiba, K
    Toyo-oka, T
    Omata, M
    Sakaki, Y
    CIRCULATION RESEARCH, 2001, 88 (02) : 202 - 209
  • [26] Atypical Ca2+-induced Ca2+ release from a sarco-endoplasmic reticulum Ca2+-ATPase 3-dependent Ca2+ pool in mouse pancreatic β-cells
    Beauvois, MC
    Arredouani, A
    Jonas, JC
    Rolland, JF
    Schuit, F
    Henquin, JC
    Gilon, P
    JOURNAL OF PHYSIOLOGY-LONDON, 2004, 559 (01): : 141 - 156
  • [27] Nitric oxide (NO) buffers intracellular Ca2+ (Ca2+i) dynamics by its opposite autocrine actions on inositol 1,4,5-trisphosphate (IP3)-induced Ca2+ release (IICR) and capacitative Ca2+ entry (CCE) in endothelial cells (ECs)
    Wang, YP
    Chen, J
    Wang, YP
    Shin, WS
    Sakamoto, A
    Nakajima, T
    Iwasawa, K
    Toyo-oka, T
    CIRCULATION, 1998, 98 (17) : 667 - 667
  • [28] In permeabilised endothelial cells IP3-induced Ca2+ release is dependent on the cytoplasmic concentration of monovalent cations (vol 37, pg 263, 1998)
    Wood, PG
    Gillespie, JI
    CARDIOVASCULAR RESEARCH, 1998, 38 (03) : 822 - 822
  • [29] Cx43 channels and signaling via IP3/Ca2+, ATP, and ROS/NO propagate radiation-induced DNA damage to non-irradiated brain microvascular endothelial cells
    Hoorelbeke, Delphine
    Decrock, Elke
    De Smet, Maarten
    De Bock, Marijke
    Descamps, Benedicte
    Van Haver, Valerie
    Delvaeye, Tinneke
    Krysko, Dmitri V.
    Vanhove, Christian
    Bultynck, Geert
    Leybaert, Luc
    CELL DEATH & DISEASE, 2020, 11 (03)
  • [30] Cx43 channels and signaling via IP3/Ca2+, ATP, and ROS/NO propagate radiation-induced DNA damage to non-irradiated brain microvascular endothelial cells
    Delphine Hoorelbeke
    Elke Decrock
    Maarten De Smet
    Marijke De Bock
    Benedicte Descamps
    Valérie Van Haver
    Tinneke Delvaeye
    Dmitri V. Krysko
    Christian Vanhove
    Geert Bultynck
    Luc Leybaert
    Cell Death & Disease, 11