TRIM72-mediated degradation of the short form of p62/SQSTM1 rheostatically controls selective autophagy in human cells

被引:11
|
作者
Wang, Cheng-Cheng [1 ,2 ]
Peng, Hong [3 ]
Wang, Zi [4 ]
Yang, Jiao [5 ]
Hu, Rong-Gui [1 ,5 ]
Li, Chuan-Yin [1 ]
Geng, Wu-Jun [6 ]
机构
[1] Tongji Univ, Shanghai Peoples Hosp 10, Sch Med, Canc Ctr, Shanghai 200040, Peoples R China
[2] Guizhou Univ, Sch Med, Guiyang 550025, Peoples R China
[3] Sun Yat Sen Univ, Sch Med, Ctr Infect & Immun Study CIIS, MOE Key Lab Trop Dis Control, Shenzhen 510275, Guangdong, Peoples R China
[4] Kyoto Univ, Inst Frontier Life & Med Sci, Kyoto 6068501, Japan
[5] Chinese Acad Sci, Shanghai Inst Biochem & Cell Biol, Ctr Excellence Mol Cell Sci, State Key Lab Mol Biol, Shanghai 200031, Peoples R China
[6] Wenzhou Med Univ, Affiliated Hosp 1, Wenzhou Key Lab Perioperat Med, Dept Anesthesiol, Wenzhou 325000, Zhejiang, Peoples R China
基金
中国国家自然科学基金;
关键词
p62S; Alternative splicing; Ubiquitination; TRIM72; Autophagy; UBIQUITYLATION;
D O I
10.1186/s40779-022-00392-1
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
引用
收藏
页数:4
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