The novel anti-cell death compound MS1 prevents ischemia-reperfusion heart injury by regulating the mitochondrial permeability transition pore

被引:0
|
作者
Sasaguri, S
Zou, ZT
Katara, RG
Asakai, R
Sodeoka, M
Enzan, H
机构
[1] Kochi Med Sch, Nankoku, Kochi, Japan
[2] Tokyo Med & Dent Univ, Tokyo, Japan
[3] Tohoku Univ, Sendai, Miyagi 980, Japan
关键词
D O I
暂无
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
1085
引用
收藏
页码:225 / 225
页数:1
相关论文
共 50 条
  • [21] A novel estrogen receptor GPER inhibits mitochondria permeability transition pore opening and protects the heart against ischemia-reperfusion injury
    Bopassa, Jean Chrisostome
    Eghbali, Mansoureh
    Toro, Ligia
    Stefani, Enrico
    AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, 2010, 298 (01): : H16 - H23
  • [22] NEUROPROTECTIVE EFFECT OF HYDROXYSAFFLOR YELLOW A AGAINST ISCHEMIA-REPERFUSION INJURY BY INHIBITING MITOCHONDRIAL PERMEABILITY TRANSITION PORE OPENING
    Ramagiri, S.
    Rajeev, T.
    JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM, 2016, 36 : 797 - 798
  • [23] Inhibition of Mitochondrial Permeability Transition Pore Opening by Activation of GPR-30 Provides Heart Protection Against Ischemia-Reperfusion Injury
    Bopassa, Jean Chrisostome
    Toro, Ligia
    Stefani, Enrico
    CIRCULATION, 2009, 120 (18) : S1462 - S1462
  • [24] SOD1 Over-Expression Prevents Mitochondrial Calcium Overload and Mitochondrial Permeability Transition in in vitro Renal Ischemia-Reperfusion Injury
    Liang, Huanling
    Sedlic, Filip
    Bosnjak, Zeljko
    Nilakantan, Vani
    FREE RADICAL BIOLOGY AND MEDICINE, 2009, 47 : S147 - S147
  • [25] DJ-1 protects the heart against ischemia-reperfusion injury by regulating mitochondrial fission
    Shimizu, Yuuki
    Lambert, Jonathan P.
    Nicholson, Chad K.
    Kim, Joshua J.
    Wolfson, David W.
    Cho, Hee Cheol
    Husain, Ahsan
    Naqvi, Nawazish
    Chin, Li-Shen
    Li, Lian
    Calvert, John W.
    JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 2016, 97 : 56 - 66
  • [26] Inhibition of Mitochondrial Permeability Transition Pore by Intralipid Protects the Late Pregnant Heart Against Ischemia Reperfusion Injury
    Li, Jingyuan
    Bopassa, Jean C.
    Umar, Soban
    Avliyakulov, Nurali
    Iorga, Andrea
    Partownavid, Parisa
    Rahman, Siamak
    Haykinson, Michael
    Eghbali, Mansoureh
    CIRCULATION, 2010, 122 (21)
  • [27] Cinnamic Anilides as New Mitochondrial Permeability Transition Pore Inhibitors Endowed with Ischemia-Reperfusion Injury Protective Effect in Vivo
    Fancelli, Daniele
    Abate, Agnese
    Amici, Raffaella
    Bernardi, Paolo
    Ballarini, Marco
    Cappa, Anna
    Carenzi, Giacomo
    Colombo, Andrea
    Contursi, Cristina
    Di Lisa, Fabio
    Dondio, Giulio
    Gagliardi, Stefania
    Milanesi, Eva
    Minucci, Saverio
    Pain, Gilles
    Pelicci, Pier Giuseppe
    Saccani, Alessandra
    Storto, Mariangela
    Thaler, Florian
    Varasi, Mario
    Villa, Manuela
    Plyte, Simon
    JOURNAL OF MEDICINAL CHEMISTRY, 2014, 57 (12) : 5333 - 5347
  • [28] Change and trend of the reperfusion injury salvage kinase and mitochondrial permeability transition pore after ischemic postconditioning in myocardial ischemia-reperfusion in rats
    Jun, Xiao
    JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY, 2014, 64 (16) : C67 - C67
  • [29] Pharmacological Postconditioning Protects Isolated Rat Hearts against Ischemia-Reperfusion Injury: The Role of Mitochondrial Permeability Transition Pore
    Duan, X.
    Yu, K.
    Ji, B. Y.
    Hei, F. L.
    Liu, J. P.
    Long, C.
    CARDIOLOGY, 2010, 117 : 94 - 94
  • [30] Transient opening of mitochondrial permeability transition pore by reactive oxygen species protects myocardium from ischemia-reperfusion injury
    Saotome, Masao
    Katoh, Hideki
    Yaguchi, Yasuhiro
    Tanaka, Takamitsu
    Urushida, Tsuyoshi
    Satoh, Hiroshi
    Hayashi, Hideharu
    AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, 2009, 296 (04): : H1125 - H1132