IGFBP3 Suppresses Reactive Oxygen Species and Cellular Senescence Response to Facilitate TGF-β-Mediated EMT

被引：0
|
作者：
Natsuizaka, Mitsuteru
Ohashi, Shinya
Ahmadi, Azal
Chang, Sanders
Nakagawa, Kei
Wong, Gabrielle S.
Miyamoto, Nobuki
Grugan, Katharine D.
Klein-Szanto, Andres J.
Herlyn, Meenhard
Diehl, J. Alan
Nakagawa, Hiroshi
机构：
来源：
GASTROENTEROLOGY | 2011年 / 140卷 / 05期
关键词：
D O I：
暂无
中图分类号：
R57 [消化系及腹部疾病];
学科分类号：
摘要：
引用
收藏
页码：S162 / S162
页数：1
相关论文
共 43 条
  • [21] Butein induces cellular senescence through reactive oxygen species-mediated p53 activation in osteosarcoma U-2 OS cells
    Hsu, Yung-Ken
    Chen, Hsuan-Ying
    Wu, Chia-Chieh
    Huang, Ying-Chih
    Hsieh, Cheng-Pu
    Su, Po-Feng
    Huang, Yi-Fu
    ENVIRONMENTAL TOXICOLOGY, 2021, 36 (05) : 773 - 781
  • [22] Defect of SIRT1-FoxO3a axis is associated with the production of reactive oxygen species during protein kinase CK2 downregulation-mediated cellular senescence and nematode aging
    Ham, Hye-Jun
    Park, Jeong-Woo
    Bae, Young-Seuk
    BMB REPORTS, 2019, 52 (04) : 265 - 270
  • [23] High glucose intake exacerbates experimental autoimmune prostatitis through mitochondrial reactive oxygen species-dependent TGF-β activation-mediated Th17 differentiation
    Niu, Di
    Yue, Shao-Yu
    Wang, Xu
    Li, Wei-Yi
    Zhang, Li
    Du, He-Xi
    Liang, Chao-Zhao
    INTERNATIONAL IMMUNOPHARMACOLOGY, 2024, 130
  • [24] MiR-29a efficiently suppresses the generation of reactive oxygen species and α-synuclein in a cellular model of Parkinson's disease by potentially targeting GSK-3β
    Yang, Ya-Ling
    Lin, Tsu-Kung
    Huang, Ying-Hsien
    EUROPEAN JOURNAL OF PHARMACOLOGY, 2024, 974
  • [25] Interleukin-35 Suppresses Endothelial Activation by Inhibiting Mitochondrial Reactive Oxygen Species Mediated Site-specific Acetylation of Histone 3 Lysine 14
    Li, Xinyuan
    Shao, Ying
    Sha, Xiaojin
    Fang, Pu
    Kuo, Yin-Ming
    Andrews, Andrew J.
    Li, Ya-feng
    Yang, William Y.
    Maddaloni, Massimo
    Pascual, David W.
    Luo, Jin J.
    Jiang, Xiaohua
    Wang, Hong
    Yang, Xiao-feng
    ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 2018, 38
  • [26] Cadmium disrupts signaling of the hypoxia-inducible (HIF) and transforming growth factor (TGF-β) pathways in placental JEG-3 trophoblast cells via reactive oxygen species
    Adebambo, Oluwadamilare A.
    Shea, Damian
    Fry, Rebecca C.
    TOXICOLOGY AND APPLIED PHARMACOLOGY, 2018, 342 : 108 - 115
  • [27] In renal proximal tubular epithelial cells of the hibernator Syrian hamster, anoxia-reoxygenation-induced reactive oxygen species bursts do not trigger a DNA damage response and cellular senescence
    Pissas, Georgios
    Divani, Maria
    Tziastoudi, Maria
    Poulianiti, Christina
    Polyzou-Konsta, Maria-Anna
    Lykotsetas, Evangelos
    Stefanidis, Ioannis
    Eleftheriadis, Theodoros
    JOURNAL OF COMPARATIVE PHYSIOLOGY B-BIOCHEMICAL SYSTEMS AND ENVIRONMENTAL PHYSIOLOGY, 2025, 195 (01): : 91 - 101
  • [28] Shikonin Derivative Suppresses Colorectal Cancer Cells Growth via Reactive Oxygen Species-Mediated Mitochondrial Apoptosis and PI3K/AKT Pathway
    Han, Hongwei
    Wen, Zhongling
    Yang, Minkai
    Wang, Changyi
    Ma, Yudi
    Chen, Qingqing
    Jiang, Dexing
    Xu, Ye
    Fazal, Aliya
    Jie, Wencai
    Lv, Xiaoran
    Yin, Tongming
    Lin, Hongyan
    Lu, Guihua
    Qi, Jinliang
    Yang, Yonghua
    Xu, Guohua
    CHEMISTRY & BIODIVERSITY, 2025,
  • [29] Sulforaphane suppresses TNF-α-mediated activation of NF-κB and induces apoptosis through activation of reactive oxygen species-dependent caspase-3
    Moon, Dong-Oh
    Kim, Mun-Ock
    Kang, Sang-Hyuck
    Choi, Yung Hyun
    Kim, Gi-Young
    CANCER LETTERS, 2009, 274 (01) : 132 - 142
  • [30] Physalin B induces apoptosis in hepatocellular carcinoma through reactive oxygen species activation via TGF-β, NF-κB and PI3K/AKT signaling pathways
    Zhao, Zhendong
    Zhou, Guohong
    Yang, Yanjun
    Xia, Li
    Xu, Dandan
    Huang, Huiqing
    Lin, Jin
    Li, Ziqian
    MATERIALS EXPRESS, 2020, 10 (04) : 578 - 584
12345