Ontogeny of Osteopontin Expression in a Non-BRICHOS Mutant Surfactant Protein C Mouse Model of Idiopathic Pulmonary Fibrosis

被引：0

作者：

Nureki, S. ^{[1
]}

Tomer, Y. ^{[2
]}

Venosa, A. ^{[2
]}

Russo, S. J. ^{[2
]}

Katzen, J. ^{[2
]}

Mulugeta, S. ^{[2
]}

Kadota, J. ^{[1
]}

Beers, M. F. ^{[2
]}

机构：

[1] Oita Univ, Fac Med, Dept Resp Med & Infect Dis, Oita, Japan

[2] Univ Penn, Sch Med, Dept Med, Div Pulm & Crit Care Med, Philadelphia, PA 19104 USA

来源：

AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE | 2018年 / 197卷

关键词：

D O I：

暂无

中图分类号：

R4 [临床医学];

学科分类号：

1002 ; 100602 ;

摘要：

A5758

引用

页数：2

共 25 条

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[23] Histone deacetylase inhibitor restores surfactant protein-C expression in alveolar-epithelial type II cells and attenuates bleomycin-induced pulmonary fibrosis in vivo
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[24] Expression of Distinct Surfactant Protein-C (SFTPC) Mutation Classes Reveal Common Downstream Pathways from Alveolar Type 2 Cell Quality Control Dysfunction to Pulmonary Fibrosis
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[25] The Relationship Between Differential Expression of Non-coding RNAs (TP53TG1, LINC00342, MALAT1, DNM3OS, miR-126-3p, miR-200a-3p, miR-18a-5p) and Protein-Coding Genes (PTEN, FOXO3) and Risk of Idiopathic Pulmonary Fibrosis
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BIOCHEMICAL GENETICS, 2025,

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