Stoichiometry and regulation network of Bcl-2 family complexes quantified by live-cell FRET assay

被引:30
|
作者
Yang, Fangfang [1 ,2 ]
Qu, Wenfeng [1 ,2 ]
Du, Mengyan [1 ,2 ]
Mai, Zihao [1 ,2 ]
Wang, Bin [1 ,2 ]
Ma, Yunyun [1 ,2 ]
Wang, Xiaoping [3 ]
Chen, Tongsheng [1 ,2 ]
机构
[1] South China Normal Univ, MOE Key Lab Laser Life Sci, Guangzhou, Peoples R China
[2] South China Normal Univ, Coll Biophoton, Guangzhou, Peoples R China
[3] Jinan Univ, Dept Pain Management, Affiliated Hosp 1, Guangzhou, Peoples R China
基金
中国国家自然科学基金;
关键词
Affinity; Bcl-2; proteins; FRET; Living cells; Stoichiometry; MEMBRANE PERMEABILIZATION; MITOCHONDRIAL-MEMBRANE; PROTEIN FAMILY; BAX; APOPTOSIS; DISTINCT; HELIX; LOCALIZATION; ACTIVATION; EFFICIENCY;
D O I
10.1007/s00018-019-03286-z
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
The stoichiometry and affinity of Bcl-2 family complexes are essential information for understanding how their interactome network is orchestrated to regulate mitochondrial permeabilization and apoptosis. Based on over-expression model system, FRET analysis was used to quantify the protein-protein interactions among Bax, Bcl-xL, Bad and tBid in healthy and apoptotic cells. Our data indicate that the stoichiometry and affinity of Bcl-2 complexes are dependent on their membrane environment. Bcl-xL, Bad and tBid can form hetero-trimers in mitochondria. Bcl-xL binds preferentially to Bad, then to tBid and Bax in mitochondria, whilst Bcl-xL displays higher affinity to Bad or tBid than to itself. Strikingly, Bax can bind to Bcl-xL in cytosol. In cytosol of apoptotic cells, Bcl-xL associates with Bax to form hetero-trimer with 1:2 stoichiometry, while Bcl-xL associates with Bad to form hetero-trimer with 2:1 stoichiometry and Bcl-xL associates with tBid to form hetero-dimer. In mitochondria, Bcl-xL associates with Bax/Bad to form hetero-dimer in healthy cells, while Bcl-xL associates with Bad to form hetero-tetramer with 3:1 stoichiometry in apoptotic cells.
引用
收藏
页码:2387 / 2406
页数:20
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