Depletion of spermine by polyamine oxidase 1 is required for nitric oxide (NO) production and associated killing of Helicobacter pylori by macrophages

被引:0
|
作者
Chaturvedi, R
Kim, PY
Potosky, D
Bussiere, FI
Asim, M
Cheng, Y
Xu, HX
Casero, RA
Wilson, KT
机构
关键词
D O I
暂无
中图分类号
R57 [消化系及腹部疾病];
学科分类号
摘要
引用
收藏
页码:A474 / A474
页数:1
相关论文
共 50 条
  • [21] Increased Helicobacter pylori-associated gastric cancer risk in the Andean region of Colombia is mediated by spermine oxidase
    R Chaturvedi
    T de Sablet
    M Asim
    M B Piazuelo
    D P Barry
    T G Verriere
    J C Sierra
    D M Hardbower
    A G Delgado
    B G Schneider
    D A Israel
    J Romero-Gallo
    T A Nagy
    D R Morgan
    T Murray-Stewart
    L E Bravo
    R M Peek
    J G Fox
    P M Woster
    R A Casero
    P Correa
    K T Wilson
    Oncogene, 2015, 34 : 3429 - 3440
  • [22] Helicobacter pylori resistant to macrophage-mediated killing are efficiently killed by macrophages deficient in NADPH oxidase activity.
    Matsumoto, Y
    Blanchard, TG
    GASTROENTEROLOGY, 2002, 122 (04) : A426 - A426
  • [23] PRODUCTION OF NITRIC-OXIDE AND SUPEROXIDE BY ACTIVATED MACROPHAGES AND KILLING OF LEISHMANIA-MAJOR
    ASSREUY, J
    CUNHA, FQ
    EPPERLEIN, M
    NORONHADUTRA, A
    ODONNELL, CA
    LIEW, FY
    MONCADA, S
    EUROPEAN JOURNAL OF IMMUNOLOGY, 1994, 24 (03) : 672 - 676
  • [24] Concomitant down-regulation of L-arginine transport and nitric oxide (NO) synthesis in rat alveolar macrophages by the polyamine spermine
    Mössner, J
    Hammermann, R
    Racké, K
    PULMONARY PHARMACOLOGY & THERAPEUTICS, 2001, 14 (04) : 297 - 305
  • [25] Spermine causes loss of innate immune response to Helicobacter pylori by inhibition of inducible nitric-oxide synthase translation
    Bussière, FI
    Chaturvedi, R
    Cheng, YL
    Gobert, AP
    Asim, M
    Blumberg, DR
    Xu, HX
    Kim, PY
    Hacker, A
    Casero, RA
    Wilson, KT
    JOURNAL OF BIOLOGICAL CHEMISTRY, 2005, 280 (04) : 2409 - 2412
  • [26] The polyamine spermine, but not putrescine, inhibits L-arginine transport activity and nitric oxide (NO) synthesis in rat alveolar macrophages (AMΦ).
    Hammermann, R
    Mössner, J
    Bliesener, N
    Wessler, I
    Racké, K
    NAUNYN-SCHMIEDEBERGS ARCHIVES OF PHARMACOLOGY, 1998, 357 (04) : R44 - R44
  • [27] LECT2 association with macrophage-mediated killing of Helicobacter pylori by activating NF-kappa B and nitric oxide production
    Shen, H. X.
    Li, L.
    Chen, Q.
    He, Y. Q.
    Yu, C. H.
    Chu, C. Q.
    Lu, X. J.
    Chen, J.
    GENETICS AND MOLECULAR RESEARCH, 2016, 15 (04)
  • [28] HELICOBACTER-PYLORI STIMULATES INDUCIBLE NITRIC-OXIDE SYNTHASE (INOS) EXPRESSION AND ACTIVITY IN MACROPHAGES
    WILSON, KT
    MUSSELMAN, RF
    MOBLEY, HLT
    JAMES, SP
    MELTZER, SJ
    RAMANUJAM, KS
    GASTROENTEROLOGY, 1995, 108 (04) : A941 - A941
  • [29] Effect of thymosin-alpha1 on the production of nitric oxide by tumor-associated macrophages
    Shrivastava, P
    Singh, SM
    Singh, N
    NEOPLASMA, 2003, 50 (01) : 47 - 53
  • [30] Helicobacter pylori arginase inhibits nitric oxide production by eukaryotic cells:: A strategy for bacterial survival
    Gobert, AP
    McGee, DJ
    Akhtar, M
    Mendz, GL
    Newton, JC
    Cheng, YL
    Mobley, HLT
    Wilson, KT
    PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2001, 98 (24) : 13844 - 13849