Hemi-specific binding by p53 expands the universe of p53 targets

被引：0
|
作者：
Vyas, P. [1 ]
Beno, I. [1 ]
Xi, Z. [2 ]
Kessler, N. [3 ]
Golovenko, D. [3 ]
Shakked, Z. [3 ]
Haran, T. E. [1 ]
机构：
[1] Technion Israel Inst Technol, Dept Biol, Haifa, Israel
[2] Yale Univ, Dept Chem, New Haven, CT USA
[3] Weizmann Inst Sci, Dept Biol Struct, Rehovot, Israel
来源：
FEBS JOURNAL | 2017年 / 284卷
关键词：
D O I：
暂无
中图分类号：
Q5 [生物化学]; Q7 [分子生物学];
学科分类号：
071010 ; 081704 ;
摘要：
P.4.1-009
引用
收藏
页码：257 / 257
页数：1
相关论文
共 50 条
  • [31] Oligomerization of BAK by p53 utilizes conserved residues of the p53 DNA binding domain
    Pietsch, E. Christine
    Perchiniak, Erin
    Canutescu, Adrian A.
    Wang, Guoli
    Dunbrack, Roland L.
    Murphy, Maureen E.
    JOURNAL OF BIOLOGICAL CHEMISTRY, 2008, 283 (30) : 21294 - 21304
  • [32] The p53 network: p53 and its downstream genes
    Shu, Kun-Xian
    Li, Biao
    Wu, Li-Xiang
    COLLOIDS AND SURFACES B-BIOINTERFACES, 2007, 55 (01) : 10 - 18
  • [33] How the p53 mRNA controls p53 activity
    Malbert-Colas, Laurence
    Tournillon, Anne-Sophie
    Olivares-Illana, Vanesa
    Punnuswainy, Anand
    Fahraeus, Robin
    INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE, 2012, 30 : S20 - S20
  • [34] Computational protein chemistry of p53 and p53 peptides
    Brandt-Rauf, PW
    Rosal, RV
    Fine, RL
    Pincus, MR
    FRONTIERS IN BIOSCIENCE-LANDMARK, 2004, 9 : 2778 - 2787
  • [35] Mutated p53 in tumors, mutant p53 and p53-specific antibodies in the circulation in patients with gastric cancer
    Maeta, M
    Saito, H
    Oka, S
    Tsujitani, S
    Ikeguchi, M
    Kaibara, N
    JOURNAL OF EXPERIMENTAL & CLINICAL CANCER RESEARCH, 2000, 19 (04) : 489 - 495
  • [36] Diverse p53/DNA binding modes expand the repertoire of p53 response elements
    Vyas, Pratik
    Beno, Itai
    Xi, Zhiqun
    Stein, Yan
    Golovenko, Dmitrij
    Kessler, Naama
    Rotter, Varda
    Shakked, Zippora
    Haran, Tali E.
    PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2017, 114 (40) : 10624 - 10629
  • [37] Modulation of alternative splicing contributes to cancer development: focusing on p53 isoforms, p53β and p53γ
    Solomon, H.
    Sharon, M.
    Rotter, V.
    CELL DEATH AND DIFFERENTIATION, 2014, 21 (09): : 1347 - 1349
  • [38] P53 α-helix mimetics antagonize p53/MDM2 interaction and activate p53
    Chen, LH
    Yin, H
    Farooqi, B
    Sebti, S
    Hamilton, AD
    Chen, JD
    MOLECULAR CANCER THERAPEUTICS, 2005, 4 (06) : 1019 - 1025
  • [39] Modulation of alternative splicing contributes to cancer development: focusing on p53 isoforms, p53β and p53γ
    H Solomon
    M Sharon
    V Rotter
    Cell Death & Differentiation, 2014, 21 : 1347 - 1349
  • [40] Protein stability of p53 targets determines their temporal expression dynamics in response to p53 pulsing
    Hanson, Ryan L.
    Porter, Joshua R.
    Batchelor, Eric
    JOURNAL OF CELL BIOLOGY, 2019, 218 (04): : 1282 - 1297
12345