MicroRNA profiling in human diploid fibroblasts uncovers miR-519 role in replicative senescence

被引:105
|
作者
Marasa, Bernard S. [1 ,2 ]
Srikantan, Subramanya [1 ]
Martindale, Jennifer L. [1 ]
Kim, Mihee M. [1 ]
Lee, Eun Kyung [1 ]
Gorospe, Myriam [1 ]
Abdelmohsen, Kotb [1 ]
机构
[1] NIA, Cellular & Mol Biol Lab, IRP, NIH, Baltimore, MD 21224 USA
[2] Catholic Univ Amer, Dept Biol, Washington, DC 20064 USA
来源
AGING-US | 2010年 / 2卷 / 06期
基金
美国国家卫生研究院;
关键词
HuR; miR-519; tumor suppression; MESSENGER-RNA; LIFE-SPAN; EXPRESSION; CELLS; HUR; MIR-34A; INHIBITION; BIOMARKER; CULTURE; CANCER;
D O I
10.18632/aging.100159
中图分类号
Q2 [细胞生物学];
学科分类号
071009 ; 090102 ;
摘要
MicroRNAs (miRNAs) are short non-coding RNAs that regulate diverse biological processes by controlling the pattern of expressed proteins. In mammalian cells, miRNAs partially complement their target sequences leading to mRNA degradation and/or decreased mRNA translation. Here, we have analyzed transcriptome-wide changes in miRNAs in senescent relative to early-passage WI-38 human diploid fibroblasts (HDFs). Among the miRNAs downregulated with senescence were members of the let-7 family, while upregulated miRNAs included miR-1204, miR-663 and miR-519. miR-519 was recently found to reduce tumor growth at least in part by lowering the abundance of the RNA-binding protein HuR. Overexpression of miR-519a in either WI-38 or human cervical carcinoma HeLa cells triggered senescence, as measured by monitoring beta-galactosidase activity and other senescence markers. These data suggest that miR-519 can suppress tumor growth by triggering senescence and that miR-519 elicits these actions by repressing HuR expression.
引用
收藏
页码:333 / 343
页数:11
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