Pneumocystis carinii β-glucan induces release of macrophage inflammatory protein-2 from primary rat alveolar epithelial cells via a receptor distinct from CD11b/CD18

被引：0
|
作者：
Hahn, PY [1 ]
Limper, AH [1 ]
机构：
[1] Mayo Clin, Thorac Dis Res Unit, Rochester, MN 55905 USA
来源：
JOURNAL OF EUKARYOTIC MICROBIOLOGY | 2001年
关键词：
D O I：
暂无
中图分类号：
Q93 [微生物学];
学科分类号：
071005 ; 100705 ;
摘要：
引用
收藏
页码：157S / 157S
页数：1
相关论文
共 38 条
  • [21] Cutting edge: Productive HIV-1 infection of dendritic cells via complement receptor type 3 (CR3, CD11b/CD18)
    Bajtay, Z
    Speth, C
    Erdei, A
    Dierich, MP
    JOURNAL OF IMMUNOLOGY, 2004, 173 (08): : 4775 - 4778
  • [22] Decreased superoxide production, degranulation, tumor necrosis factor alpha secretion, and CD11b/CD18 receptor expression by adherent monocytes from preterm infants
    Kaufman, D
    Kilpatrick, L
    Hudson, RG
    Campbell, DE
    Kaufman, A
    Douglas, SD
    Harris, MC
    CLINICAL AND DIAGNOSTIC LABORATORY IMMUNOLOGY, 1999, 6 (04) : 525 - 529
  • [23] Pneumocystis carinii B-glucan-induced chemokine secretion from alveolar epithelial cells requires activation of protein kinase C and NF-KB
    Evans, SE
    Hahn, PY
    Lebron-Ruiz, FM
    Kottom, TJ
    Puri, V
    Pagano, RE
    Limper, AH
    CHEST, 2003, 124 (04) : 188S - 188S
  • [24] Urokinase-type plasminogen activator receptor reversibly dissociates from complement receptor type 3 (alpha(M)beta(2')CD11b/CD18) during neutrophil polarization
    Kindzelskii, AL
    Laska, ZO
    Todd, RF
    Petty, HR
    JOURNAL OF IMMUNOLOGY, 1996, 156 (01): : 297 - 309
  • [25] Tumor necrosis factor-α mediates lipopolysaccharide-induced macrophage inflammatory protein-2 release from alveolar epithelial cells -: Autoregulation in host defense
    Xavier, AM
    Isowa, N
    Cai, L
    Dziak, E
    Opas, M
    McRitchie, DI
    Slutsky, AS
    Keshavjee, SH
    Liu, MY
    AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY, 1999, 21 (04) : 510 - 520
  • [26] Targeting of natural killer cells to mammary carcinoma via naturally occurring tumor cell-bound iC3b and beta-glucan-primed CR3 (CD11b/CD18)
    Vetvicka, V
    Thornton, BP
    Wieman, TJ
    Ross, GD
    JOURNAL OF IMMUNOLOGY, 1997, 159 (02): : 599 - 605
  • [27] Targeting of murine macrophages and NK cells to mammary carcinomas via naturally occurring tumor cell-bound iC3b and beta-glucan-primed CR3 (CD11b/CD18).
    Ross, GD
    Vetvicka, V
    JOURNAL OF LEUKOCYTE BIOLOGY, 1996, : 67 - 67
  • [28] Increased respiratory burst and increased expression of complement receptor-3 (CD11b/CD18) and of IL-8 receptor-A in neutrophil granulocytes from newborns after vaginal delivery
    Gessler, P
    Dahinden, C
    BIOLOGY OF THE NEONATE, 2003, 83 (02): : 107 - 112
  • [29] Reversible inactivation of purified leukocyte integrin CR3 (CD11b/CD18, alpha(m)beta(2)) by removal of divalent cations from a cryptic site
    Cai, TQ
    Law, SKA
    Zhao, HR
    Wright, SD
    CELL ADHESION AND COMMUNICATION, 1995, 3 (05) : 399 - 406
  • [30] The β-glucan-binding lectin site of mouse CR3 (CD11b/CD18) and its function in generating a primed state of the receptor that mediates cytotoxic activation in response to iC3b-opsonized target cells
    Xia, Y
    Vetvicka, V
    Yan, J
    Hanikyrová, M
    Mayadas, T
    Ross, GD
    JOURNAL OF IMMUNOLOGY, 1999, 162 (04): : 2281 - 2290
1234