Hybrid Amyloid Fibril Genesis with Components of Sporadic and Familial Alzheimer's Disease

被引：0
|
作者：
Ollesch, Julian [1 ]
Faendrich, Marcus [1 ]
机构：
[1] Max Planck Res Unit Enzymol Prot Folding, Halle, Saale, Germany
来源：
BIOPHYSICAL JOURNAL | 2010年 / 98卷 / 03期
关键词：
D O I：
10.1016/j.bpj.2009.12.1385
中图分类号：
Q6 [生物物理学];
学科分类号：
071011 ;
摘要：
引用
收藏
页码：255A / 255A
页数：1
相关论文
共 50 条
  • [31] The association of circulating amylin with β-amyloid in familial Alzheimer's disease
    Ly, Han
    Verma, Nirmal
    Sharma, Savita
    Kotiya, Deepak
    Despa, Sanda
    Abner, Erin L.
    Nelson, Peter T.
    Jicha, Gregory A.
    Wilcock, Donna M.
    Goldstein, Larry B.
    Guerreiro, Rita
    Bras, Jose
    Hanson, Angela J.
    Craft, Suzanne
    Murray, Andrew J.
    Biessels, Geert Jan
    Troakes, Claire
    Zetterberg, Henrik
    Hardy, John
    Lashley, Tammaryn
    Despa, Florin
    ALZHEIMERS & DEMENTIA-TRANSLATIONAL RESEARCH & CLINICAL INTERVENTIONS, 2021, 7 (01)
  • [32] Amyloid imaging in presymptomatic and symptomatic familial Alzheimer's disease
    Johnson, KA
    Rentz, D
    Moran, EK
    Becker, JA
    Sperling, R
    Pollen, D
    Moonis, M
    Swearer, J
    Hyman, B
    Selkoe, D
    DeKosky, S
    Mathis, CA
    Munk, WE
    Fischman, AJ
    NEUROLOGY, 2006, 66 (05) : A102 - A102
  • [33] The role of lipid membrane in amyloid fibril formation and toxicity in Alzheimer's disease
    Drolle, E.
    Hane, F.
    Choi, Y.
    Attwood, S.
    Ollagnier, A.
    Finot, E.
    Leonenko, Z.
    EUROPEAN BIOPHYSICS JOURNAL WITH BIOPHYSICS LETTERS, 2013, 42 : S120 - S120
  • [34] Etiology of sporadic Alzheimer's disease:: Somatostatin, neprilysin, and amyloid β peptide
    Hama, E
    Saido, TC
    MEDICAL HYPOTHESES, 2005, 65 (03) : 498 - 500
  • [35] Update on amyloid- homeostasis markers for sporadic Alzheimer's disease
    Zetterberg, Henrik
    SCANDINAVIAN JOURNAL OF CLINICAL & LABORATORY INVESTIGATION, 2009, 69 (01): : 18 - 21
  • [36] Cross-seeding of WT amyloid-β with Arctic but not Italian familial mutants accelerates fibril formation in Alzheimer's disease
    Liang, Ruina
    Tian, Yao
    Viles, John H.
    JOURNAL OF BIOLOGICAL CHEMISTRY, 2022, 298 (07)
  • [37] Amyloid-β40 E22K fibril in familial Alzheimer's disease is more thermostable and susceptible to seeding
    Lee, Ming-Che
    Liao, Yi-Hung
    Chen, Shih-Hui
    Chen, Yun-Ru
    IUBMB LIFE, 2022, 74 (08) : 739 - 747
  • [38] Cathepsin D polymorphism in Italian sporadic and familial Alzheimer's disease
    Bagnoli, S
    Nacmias, B
    Tedde, A
    Guarnieri, BM
    Cellini, E
    Ciantelli, M
    Petruzzi, C
    Bartoli, A
    Ortenzi, L
    Serio, A
    Sorbi, S
    NEUROSCIENCE LETTERS, 2002, 328 (03) : 273 - 276
  • [39] Tau load is associated with apolipoprotein E genotype and the amount of amyloid β protein, Aβ40, in sporadic and familial Alzheimer's disease
    Thaker, U
    McDonagh, AM
    Iwatsubo, T
    Lendon, CL
    Pickering-Brown, SM
    Mann, DMA
    NEUROPATHOLOGY AND APPLIED NEUROBIOLOGY, 2003, 29 (01) : 35 - 44
  • [40] Auditory adaptation is differentially impaired in familial and sporadic Alzheimer's disease
    Tarkka, IM
    Lehtovirta, M
    Soininen, H
    Pääkkönen, A
    Karhu, J
    Partanen, J
    BIOMEDICINE & PHARMACOTHERAPY, 2002, 56 (01) : 45 - 49
12345