In vivo Tissue Cholesterol Efflux is Impaired in Carriers of Mutations in APOA1 and ABCA1

被引：0
|
作者：
Holleboom, Adriaan G.
Franssen, Remco
Jakulj, Lily
Decaris, Julie
Vergeer, Menno
Koetsveld, Joris
Hellerstein, Marc K.
Kastelein, John J.
Groen, Albert K.
Turner, Scott M.
Stroes, Erik S.
机构：
[1] Acad Med Cntr, Amsterdam, Netherlands
[2] Kinemed Inc, Emeryville, CA USA
[3] Univ Calif San Francisco, San Francisco, CA 94143 USA
[4] Univ Groningen, Groningen, Netherlands
来源：
CIRCULATION | 2010年 / 122卷 / 21期
关键词：
HDL; Gene mutations; Lipoproteins;
D O I：
暂无
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
A17881
引用
收藏
页数：2
相关论文
共 50 条
  • [41] ABCA1 and ABCG1 synergize to mediate cholesterol export to apoA-I
    Gelissen, IC
    Harris, M
    Rye, KA
    Quinn, C
    Brown, AJ
    Kockx, M
    Cartland, S
    Packianathan, M
    Kritharides, L
    Jessup, W
    ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 2006, 26 (03) : 534 - 540
  • [42] ATVB in focus - Role of ABCA1 in cellular cholesterol efflux and reverse cholesterol transport
    Tall, AR
    ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 2003, 23 (05) : 710 - 711
  • [43] Increased ABCA1-mediated cholesterol efflux potential of sera from APOA-I Milano carriers
    Favari, E
    Zanotti, I
    Bernini, F
    Lee, M
    Kovanen, PT
    Sirtori, CR
    Franceschini, G
    Calabresi, L
    ATHEROSCLEROSIS SUPPLEMENTS, 2005, 6 (01) : 26 - 26
  • [44] A PEST deletion mutant of ABCA1 shows impaired internalization and defective cholesterol efflux from late endosomes
    Chen, WG
    Wang, N
    Tall, AR
    JOURNAL OF BIOLOGICAL CHEMISTRY, 2005, 280 (32) : 29277 - 29281
  • [45] GENETIC ASSOCIATION OF SINGLE NUCLEOTIDE POLYMORPHISMS OF ABCA1, APOA1 AND LCAT GENES IN SUBJECTS WITH LOW CONCENTRATION OF HIGH DENSITY LIPOPROTEIN-CHOLESTEROL
    Sakri, F. H.
    Mokhsin, N. A. Mohd
    Ahmad, W. N. H. Wan
    Rahman, T. Abdul
    Hoh, B. P.
    Nawawi, H. Mohd
    ATHEROSCLEROSIS, 2015, 241 (01) : E109 - E109
  • [46] Cytotoxic cellular cholesterol is selectively removed by apoA-I via ABCA1
    Kellner-Weibel, G
    Luke, SJ
    Rothblat, GH
    ATHEROSCLEROSIS, 2003, 171 (02) : 235 - 243
  • [47] ABCA1 C-terminal mutations reveal a novel VFVNFA motif that is required for cholesterol efflux activity
    Fitzgerald, ML
    Okuhira, K
    Manning, JJ
    Bell, SA
    Freeman, MW
    ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 2004, 24 (05) : E1 - E1
  • [48] Hepatobiliary cholesterol transport is not impaired in ABCA1 null mice.
    Groen, AK
    Bloks, V
    Brandsma, R
    Ottenhoff, R
    Chimini, G
    Kuipers, F
    GASTROENTEROLOGY, 2001, 120 (05) : A550 - A550
  • [49] Characterization of apoA-I-dependent lipid efflux from adipocytes and role of ABCA1
    Howard, Alisha D.
    Verghese, Philip B.
    Arrese, Estela L.
    Soulages, Jose L.
    MOLECULAR AND CELLULAR BIOCHEMISTRY, 2010, 343 (1-2) : 115 - 124
  • [50] Characterization of apoA-I-dependent lipid efflux from adipocytes and role of ABCA1
    Alisha D. Howard
    Philip B. Verghese
    Estela L. Arrese
    Jose L. Soulages
    Molecular and Cellular Biochemistry, 2010, 343 : 115 - 124
12345