Paraoxonase-1 Q192R polymorphism and risk of sporadic amyotrophic lateral sclerosis

被引：8
|
作者：
Slowik, A
Tomik, B
Partyka, D
Turaj, W
Pera, J
Dziedzic, T
Szermer, P
Figlewicz, DA
Szczudlik, A
机构：
[1] Jagiellonian Univ, Dept Neurol, PL-31503 Krakow, Poland
[2] Univ Michigan, Dept Neurol, Ann Arbor, MI 48109 USA
来源：
CLINICAL GENETICS | 2006年 / 69卷 / 04期
关键词：
D O I：
10.1111/j.1399-0004.2006.00590.x
中图分类号：
Q3 [遗传学];
学科分类号：
071007 ; 090102 ;
摘要：
引用
收藏
页码：358 / 359
页数：2
相关论文
共 50 条
  • [21] Association of the Paraoxonase 1 Q192R Genetic Polymorphism with Disease Activity in Dermatomyositis
    Bae, Sangmee
    Oganesian, Buzand
    Dowd, Tyler
    Golub, Ilana
    Shahbazian, Ani
    Wang, Jennifer
    Reddy, Srinivasa T.
    Charles-Schoeman, Christina
    ARTHRITIS & RHEUMATOLOGY, 2017, 69
  • [22] Cholinesterases activity and paraoxonase 1 Q192R polymorphism of workers exposed to pesticides
    Molina-Pintor, I. B.
    Bernal-Hernandez, Y. Y.
    Rojas-Garcia, A. E.
    Medina-Diaz, I. M.
    Herrera Moreno, J. F.
    Alvarez Puentes, G. A.
    Barron-Vivanco, B. S.
    TOXICOLOGY LETTERS, 2016, 259 : S210 - S210
  • [23] Investigating paraoxonase-1 gene Q192R and L55M polymorphism in patients with renal cell cancer
    Uyar, O. A.
    Kara, M.
    Erol, D.
    Ardicoglu, A.
    Yuce, H.
    GENETICS AND MOLECULAR RESEARCH, 2011, 10 (01) : 133 - 139
  • [24] The Q192R polymorphism of the paraoxonase-1 (PON1) gene is associated with susceptibility to gestational diabetes mellitus in the Greek population
    Pappa, Kalliopi I.
    Gazouli, Maria
    Anastasiou, Eleni
    Loutradis, Dimitrios
    Anagnou, Nicholas P.
    GYNECOLOGICAL ENDOCRINOLOGY, 2017, 33 (08) : 617 - 620
  • [25] The Q192R polymorphism of the paraoxonase 1 gene is a risk factor for coronary artery disease in Saudi subjects
    Mohammed A. Hassan
    Omar S. Al-Attas
    Tajamul Hussain
    Nasser M. Al-Daghri
    Majed S. Alokail
    Abdul K. Mohammed
    Benjamin Vinodson
    Molecular and Cellular Biochemistry, 2013, 380 : 121 - 128
  • [26] The Q192R polymorphism of the paraoxonase 1 gene is a risk factor for coronary artery disease in Saudi subjects
    Hassan, Mohammed A.
    Al-Attas, Omar S.
    Hussain, Tajamul
    Al-Daghri, Nasser M.
    Alokail, Majed S.
    Mohammed, Abdul K.
    Vinodson, Benjamin
    MOLECULAR AND CELLULAR BIOCHEMISTRY, 2013, 380 (1-2) : 121 - 128
  • [27] Paraoxonase-1 Q192R polymorphism and its association with hs-CRP and fasting blood glucose levels and risk of coronary artery disease
    Amini, Mahsa
    Esmaeilzadeh-bahabadi, Sedigheh
    Avan, Amir
    Gholoobi, Aida
    Ghasemi, Faezeh
    Mirhafez, Seyed Reza
    Ghazizadeh, Hamideh
    Moohebati, Mohsen
    Ebrahimi, Mahmoud
    Ferns, Gordon A.
    Pasdar, Alireza
    Mobarhan, Majid Ghayour
    DIABETES & METABOLIC SYNDROME-CLINICAL RESEARCH & REVIEWS, 2019, 13 (02) : 1053 - 1057
  • [28] Association of L55M and Q192R Polymorphisms of Paraoxonase-1 Gene with Preeclampsia
    Yaghmaei, Minoo
    Hashemi, Mohammad
    Azarian, Azin
    Moazeni-Roodi, Abdolkarim
    Mokhtari, Mojgan
    Naghavai, Anoosh
    Salimi, Saeideh
    Mohammadi, Mandi
    Taheri, Mohsen
    Ghavami, Saeid
    ARCHIVES OF MEDICAL RESEARCH, 2011, 42 (04) : 324 - 328
  • [29] PON1 is a disease modifier gene in amyotrophic lateral sclerosis: association of the Q192R polymorphism with bulbar onset and reduced survival
    Federico Verde
    Cinzia Tiloca
    Claudia Morelli
    Alberto Doretti
    Barbara Poletti
    Luca Maderna
    Stefano Messina
    Davide Gentilini
    Isabella Fogh
    Antonia Ratti
    Vincenzo Silani
    Nicola Ticozzi
    Neurological Sciences, 2019, 40 : 1469 - 1473
  • [30] PON1 is a disease modifier gene in amyotrophic lateral sclerosis: association of the Q192R polymorphism with bulbar onset and reduced survival
    Verde, Federico
    Tiloca, Cinzia
    Morelli, Claudia
    Doretti, Alberto
    Poletti, Barbara
    Maderna, Luca
    Messina, Stefano
    Gentilini, Davide
    Fogh, Isabella
    Ratti, Antonia
    Silani, Vincenzo
    Ticozzi, Nicola
    NEUROLOGICAL SCIENCES, 2019, 40 (07) : 1469 - 1473
12345