Cathepsin D expressed in hepatocytes does not participate in the development of liver fibrosis after chronic CCl4 administration

被引：0

作者：

Ruiz-Blazquez, Paloma ^{[1
,2
]}

Fernandez-Fernandez, Maria ^{[1
,2
]}

Pistorio, Valeria ^{[3
,4
]}

Nunez, Susana ^{[2
]}

Carmen Garcia-Ruiz, M. ^{[1
,2
,5
,6
]}

Fernandez-Checa, Jose ^{[1
,2
,5
,6
]}

Moles, Anna ^{[1
,2
,5
]}

机构：

[1] Inst Invest Biomed Barcelona IIBB CSIC, Barcelona, Spain

[2] Ctr Invest Biomed Red Enfermedades Hepat & Digest, Barcelona, Spain

[3] Sorbonne Univ, CRSA, INSERM, Paris, France

[4] Univ Naples Federico II, Naples, Italy

[5] Inst Invest Biomed August Pi & Sunyer IDIBAPS, Barcelona, Spain

[6] USC Res Ctr ALPD, Los Angeles, CA USA

来源：

JOURNAL OF HEPATOLOGY | 2023年 / 78卷

关键词：

D O I：

暂无

中图分类号：

R57 [消化系及腹部疾病];

学科分类号：

摘要：

WED-247

引用

页码：S339 / S339

页数：1

共 43 条

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[33] Co-administration of resveratrol and beta-aminopropionitrile attenuates liver fibrosis development via targeting lysyl oxidase in CCl4-induced liver fibrosis in rats
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[34] ISOENZYMES OF N-ACETYL-BETA-D-GLUCOSAMINIDASE IN LIVER-TISSUE AND BLOOD OF NORMAL RATS AND AFTER POISONING WITH CCL4
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[35] Resolvin D1 attenuates CCl4 Induced Liver Fibrosis by Inhibiting Autophagy-Mediated HSC activation via AKT/mTOR Pathway
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[36] The influence of Xymedon conjugate with L-ascorbic acid on initial development of fibrosis in the rat liver after toxic exposure of CCl 4
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Belyaev, G.
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EUROPEAN JOURNAL OF CLINICAL INVESTIGATION, 2021, 51 : 78 - 78
[37] DOSE-DEPENDENT CONTRIBUTION OF HUMAN PERIPHERAL BLOOD CD34-POSITIVE CELL TRANSPLANTATION TO HEPATIC REGENERATION AFTER CCL4 CHRONIC LIVER INJURY
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[38] Decrease of platelet-endothelial cell adhesion - Gene expression in inflammatory cells and in endothelial cells in the rat liver following CCL4 administration and in vitro after treatment with TNF-ALPHA.
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[39] Alleviation of liver damage and hepatic fibrosis by oral administration of Imm 124E colostrums in Carbon tetra-chloride (CCl4) model is mediated by decrease of intra-hepatic F4/80 macrophages activation
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HEPATOLOGY, 2013, 58 : 588A - 589A
[40] Recipient CYP2D6*4 Poor Metabolizer Status Associates with Early Fibrosis Development after Liver Transplantation
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