Sickle red blood cell adherence to endothelium is stronger for attachment under dynamic shear flow

被引：0

作者：

McNaull, Stewart ^{[1
]}

Eckman, James ^{[1
]}

Hsu, Lewis ^{[1
]}

Wick, Timothy ^{[1
]}

机构：

[1] Bayer Corp, Clayton, United States

来源：

Annals of Biomedical Engineering | 2000年 / 28卷 / SUPPL. 1期

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摘要：

引用

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[1] Sickle red blood cell adherence to endothelium is modulated by hypoxia
Setty, BNY
Stuart, MJ
THROMBOSIS AND HAEMOSTASIS, 1997, : P1868 - P1868
[2] Sickle red blood cell adherence to vascular endothelium is more tenacious in confined flow channels
McNaull, SA
Eckman, JR
Wick, TM
BLOOD, 1996, 88 (10) : 23 - 23
[3] Inflammatory mediators promote sickle red blood cell adhesion to endothelium under venular flow
Owings, A
Wagner, M
Wick, T
PROCEEDINGS OF THE 23RD EUROPEAN CONFERENCE ON MICROCIRCULATION, 2004, : 33 - 38
[4] Nitric oxide attenuates normal and sickle red blood cell adherence to pulmonary endothelium
Space, SL
Lane, PA
Pickett, CK
Weil, JV
AMERICAN JOURNAL OF HEMATOLOGY, 2000, 63 (04) : 200 - 204
[5] Inflammatory mediators promote strong sickle cell adherence to endothelium under venular flow conditions
Walmet, PS
Eckman, JR
Wick, TM
AMERICAN JOURNAL OF HEMATOLOGY, 2003, 73 (04) : 215 - 224
[6] Low blood oxygen saturation levels are associated with increased sickle red blood cell (RBC) adherence to endothelium.
Setty, BNY
Dampier, C
Chen, D
ONeal, P
Reber, D
Allen, J
Stuart, M
BLOOD, 1996, 88 (10) : 25 - 25
[7] Structural Analysis of Red Blood Cell Aggregates Under Shear Flow
Chesnutt, J. K. W.
Marshall, J. S.
ANNALS OF BIOMEDICAL ENGINEERING, 2010, 38 (03) : 714 - 728
[8] Structural Analysis of Red Blood Cell Aggregates Under Shear Flow
J. K. W. Chesnutt
J. S. Marshall
Annals of Biomedical Engineering, 2010, 38 : 714 - 728
[9] Shear stress modulation of cytokine-induced sickle cell adherence to endothelium.
Wick, TM
Wagner, MC
Eckman, JR
BLOOD, 2003, 102 (11) : 264A - 264A
[10] Tenacity of sickle red blood cell (SRBC) endothelial cell (EC) adherence is augmented under hemodynamic shear and by involvement of multiple adhesion pathways.
Smolinski, PA
Eckman, JR
Wick, TM
BLOOD, 1996, 88 (10) : 2585 - 2585

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