CENTRALLY MEDIATED EFFECTS OF CARDIAC DRUGS - STROPHANTHIN-K, QUINIDINE AND PROCAINE AMIDE

被引：39

|

作者：

WEINBERG, SJ

HALEY, TJ

机构：

来源：

CIRCULATION RESEARCH | 1955年 / 3卷 / 01期

关键词：

D O I：

10.1161/01.RES.3.1.103

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

收藏

页码：103 / 109

页数：7

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[21] Effects of centrally-acting antihypertensive drugs in a model of isoprenaline-induced cardiac hypertrophy
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[22] STUDIES BY ELECTRON MICROSCOPY ON EFFECTS OF MAGNESIUM CHLORIDE-PROCAINE AMIDE OR POTASSIUM CITRATE ON MYOCARDIUM IN INDUCED CARDIAC ARREST
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[23] EFFECTS OF ANTIARRHYTHMIC AND BRADYCARDIAC DRUGS ON CLONED CARDIAC K+ CHANNELS
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ISHII, K
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[24] ROLE OF NA+,K+-ATPASE IN THE CENTRALLY MEDIATED HYPOTENSIVE EFFECTS OF POTASSIUM IN ANESTHETIZED RATS
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[25] EFFECTS OF PH UPON THE INHIBITION BY SULFONYLUREA DRUGS OF ATP-SENSITIVE K+ CHANNELS IN CARDIAC-MUSCLE
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[28] ENDOTHELIN-1 (ET-1) INHIBITS CENTRALLY MEDIATED HYPOTENSIVE AND BRADYCARDIC EFFECTS OF KCL IN ANESTHETIZED RATS - A OUABAIN-LIKE ACTION ON THE NEURONAL NA+,K+- ATPASE
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[30] Interplay between the effects of a Protein Kinase C phosphomimic (T204E) and a dilated cardiomyopathy mutation (K211Δ or R206W) in rat cardiac troponin T blunts the magnitude of muscle length-mediated crossbridge recruitment against the β-myosin heavy chain background
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