EPSTEIN-BARR-VIRUS BINDING INDUCES INTERNALIZATION OF THE C3D RECEPTOR - A NOVEL IMMUNOTOXIN DELIVERY SYSTEM

被引：0

作者：

TEDDER, TF ^{[1
]}

GOLDMACHER, VS ^{[1
]}

LAMBERT, JM ^{[1
]}

SCHLOSSMAN, SF ^{[1
]}

机构：

[1] HARVARD UNIV,SCH MED,DEPT MED,BOSTON,MA 02115

来源：

JOURNAL OF IMMUNOLOGY | 1986年 / 137卷 / 04期

关键词：

D O I：

暂无

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

100102 ;

摘要：

引用

页码：1387 / 1391

页数：5

共 50 条

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[32] SUPERINFECTION OF EPITHELIAL HYBRID-CELLS (D98/HR-1, NPC-KT, AND A2L/AH) WITH EPSTEIN-BARR-VIRUS AND THE RELATIONSHIP TO THE C3D RECEPTOR
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[33] BIOCHEMICAL, ANTIGENIC AND FUNCTIONAL-PROPERTIES OF THE PURIFIED EPSTEIN-BARR-VIRUS C3D-RECEPTOR OF HUMAN LYMPHOCYTES-B
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[35] IDENTIFICATION AND CHARACTERIZATION OF THE EPSTEIN-BARR VIRUS RECEPTOR ON HUMAN LYMPHOCYTES-B AND ITS RELATIONSHIP TO THE C3D COMPLEMENT RECEPTOR (CR-2)
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[36] A COMPLEMENT RECEPTOR LOCUS - GENES ENCODING C3B/C4B RECEPTOR AND C3D/EPSTEIN-BARR VIRUS RECEPTOR MAP TO 1Q32
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[37] C3D AND EPSTEIN-BARR-VIRUS (CR2/CD21 LIGANDS) STIMULATE CELLS OF AN HTLV-I LINE, MT-2
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[38] EXPRESSION OF FUNCTIONAL HUMAN EPSTEIN-BARR-VIRUS C3D RECEPTOR ([CR-2] CD21) ON INSULINOMA CELL-LINE - INDUCTION OF TUMOR REJECTION BUT NOT DIABETES IN SYNGENEIC RATS
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[39] EPSTEIN-BARR-VIRUS C3D RECEPTOR (CR2, CD21) ACTIVATED BY ITS EXTRACELLULAR LIGANDS REGULATES PP105 PHOSPHORYLATION THROUGH 2 DISTINCT PATHWAYS
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[40] IDENTIFICATION OF A HUMAN EPITHELIAL-CELL SURFACE PROTEIN SHARING AN EPITOPE WITH THE C3D/EPSTEIN-BARR VIRUS RECEPTOR MOLECULE OF B LYMPHOCYTES
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