Deletion of the RNA-binding proteins ZFP36L1 and ZFP36L2 leads to perturbed thymic development and T lymphoblastic leukemia

被引:0
|
作者
Daniel J Hodson
Michelle L Janas
Alison Galloway
Sarah E Bell
Simon Andrews
Cheuk M Li
Richard Pannell
Christian W Siebel
H Robson MacDonald
Kim De Keersmaecker
Adolfo A Ferrando
Gerald Grutz
Martin Turner
机构
[1] Laboratory of Lymphocyte Signalling and Development,Department of Molecular Biology
[2] The Babraham Institute,Department of Pathology
[3] Bioinformatics Group,Department of Pediatrics
[4] The Babraham Institute,undefined
[5] Medical Research Council Laboratory of Molecular Biology,undefined
[6] Genentech,undefined
[7] Ludwig Institute for Cancer Research,undefined
[8] Lausanne Branch,undefined
[9] University of Lausanne,undefined
[10] Institute for Cancer Genetics,undefined
[11] Columbia University Medical Centre,undefined
[12] Columbia University Medical Centre,undefined
[13] Columbia University Medical Centre,undefined
[14] Institute of Medical Immunology,undefined
[15] Charité-Universitätsmedizin,undefined
[16] Campus Mitte,undefined
[17] Center for Biomaterial Development,undefined
[18] Institute of Polymer Research,undefined
[19] GKSS,undefined
来源
Nature Immunology | 2010年 / 11卷
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摘要
RNA-binding proteins are involved in post-transcriptional regulation. Turner and co-workers show that these proteins are also critical in thymopoiesis.
引用
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页码:717 / 724
页数:7
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