Low levels of apolipoprotein B in heterozygous hypobetalipoproteinemic knock out mice expressing truncated ApoB species occur by low production of ApoB100 but not B48

被引：0
|
作者：
Srivastava, RAK
Srivastava, N
Toth, L
Maeda, N
Cefalu, AB
Averna, MR
Schonfeld, G
机构：
[1] WASHINGTON UNIV,ST LOUIS,MO
[2] UNIV N CAROLINA,CHAPEL HILL,NC
[3] UNIV PALERMO,PALERMO,ITALY
来源：
CIRCULATION | 1997年 / 96卷 / 08期
关键词：
D O I：
暂无
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
引用
收藏
页码：3040 / 3040
页数：2
相关论文
共 26 条
  • [1] Regulation of the apolipoprotein B in heterozygous hypobetalipoproteinemic knock-out mice expressing truncated apoB, B81. Low production and enhanced clearance of apoB cause low levels of apoB
    Srivastava, RAK
    Toth, L
    Srivastava, N
    Hinsdale, ME
    Maeda, N
    Cefalu, AB
    Averna, M
    Schonfeld, G
    MOLECULAR AND CELLULAR BIOCHEMISTRY, 1999, 202 (1-2) : 37 - 46
  • [2] Regulation of the apolipoprotein B in heterozygous hypobeta-lipoproteinemic knock-out mice expressing truncated apoB, B81. Low production and enhanced clearance of apoB cause low levels of apoB
    Rai Ajit K. Srivastava
    L. Toth
    Neelam Srivastava
    Myron E. Hinsdale
    Nobuyo Maeda
    A.B. Cefalu´
    Maurizio Averna
    Gustav Schonfeld
    Molecular and Cellular Biochemistry, 1999, 202 : 37 - 46
  • [3] Phenotypic analysis of mice expressing exclusively apolipoprotein B48 or apolipoprotein B100
    Farese, RV
    Veniant, MM
    Cham, CM
    Flynn, LM
    Pierotti, V
    Loring, JF
    Traber, M
    Ruland, S
    Stokowski, RS
    Huszar, D
    Young, SG
    PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1996, 93 (13) : 6393 - 6398
  • [4] LINOLEIC-ACID IS A VERY POTENT APOLIPOPROTEIN B48/APOB100 SECRETAGOGUE IN POLARIZED CACO-2 CELL MONOLAYERS
    DEBRUIN, TWA
    VANGREEVENBROEK, M
    BOURITIUS, H
    VAN MEER, G
    ERKELENS, DW
    CIRCULATION, 1992, 86 (04) : 748 - 748
  • [5] Susceptibility to atherosclerosis in mice expressing exclusively apolipoprotein B48 or apolipoprotein B100
    Veniant, MM
    Pierotti, V
    Newland, D
    Cham, CM
    Sanan, DA
    Walzem, RL
    Young, SG
    JOURNAL OF CLINICAL INVESTIGATION, 1997, 100 (01): : 180 - 188
  • [6] Normal intestinal dietary fat and cholesterol absorption, intestinal apolipoprotein B (ApoB) mRNA levels, and ApoB-48 synthesis in a hypobetalipoproteinemic kindred without any ApoB truncation
    Pulai, JI
    Averna, M
    Srivastava, RAK
    Latour, MA
    Clouse, RE
    Ostlund, RE
    Schonfeld, G
    METABOLISM-CLINICAL AND EXPERIMENTAL, 1997, 46 (09): : 1095 - 1100
  • [7] A FRAMESHIFT MUTATION IN THE APO-B GENE YIELDS A TRUNCATED APO-B SPECIES, APOB86, AND A FULL-LENGTH APOB100
    LINTON, MF
    PIEROTTI, VR
    YOUNG, SG
    CIRCULATION, 1992, 86 (04) : 209 - 209
  • [8] A novel inhibitor of oxidosqualene: Lanosterol cyclase inhibits very low-density lipoprotein apolipoprotein B100 (ApoB100) production and enhances low-density lipoprotein ApoB100 catabolism through marked reduction in hepatic cholesterol content
    Telford, DE
    Lipson, SM
    Barrett, PHR
    Sutherland, BG
    Edwards, JY
    Aebi, JD
    Dehmlow, H
    Morand, OH
    Huff, MW
    ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 2005, 25 (12) : 2608 - 2614
  • [9] Liraglutide Reduces Postprandial Hyperlipidemia by Increasing ApoB48 (Apolipoprotein B48) Catabolism and by Reducing ApoB48 Production in Patients With Type 2 Diabetes Mellitus
    Verges, Bruno
    Duvillard, Laurence
    de Barros, Jean Paul Pais
    Bouillet, Benjamin
    Baillot-Rudoni, Sabine
    Rouland, Alexia
    Sberna, Anne-Laure
    Petit, Jean-Michel
    Degrace, Pascal
    Demizieux, Laurent
    ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 2018, 38 (09) : 2198 - 2206
  • [10] Molecular bases of low production rates of apolipoprotein B-100 and truncated apoB-82 in a mutant HepG2 cell line generated by targeted modification of the apolipoprotein B gene
    Srivastava, RAK
    Srivastava, N
    Averna, M
    Cefalu, AB
    Schonfeld, G
    JOURNAL OF LIPID RESEARCH, 1999, 40 (05) : 901 - 912
123