Myeloid metaplasia with myelofibrosis: Cytogenetic and FISH demonstration that CD34+cells but not fibroblasts belong to the pathological clone.

被引：0
|
作者：
Bilhou-Nabera, C
Brigaudeau, C
Clay, D
Lai, JL
Brouty-Boye, D
Vignon, C
Gharbi, MJ
Le Bousse-Kerdiles, MC
Praloran, V
机构：
[1] Univ Bordeaux 2, Hematol Lab, F-33076 Bordeaux, France
[2] Hop Paul Brousse, INSERM U268, Villejuif, France
[3] Dupuytren Hosp, Hematol Lab, Limoges, France
[4] Jeanne de Flandres Hosp, Med Genet Lab, Lille, France
来源：
BLOOD | 2002年 / 100卷 / 11期
关键词：
D O I：
暂无
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
2316
引用
收藏
页码：589A / 589A
页数：1
相关论文
共 27 条
  • [1] Marrow Fibrosis Does Not Account for Circulating CD34+Cells in Myelofibrosis with Myeloid Metaplasia
    Samuelson, Scott J.
    Prchal, Josef T.
    HAEMATOLOGICA, 2007, 92 (04) : 47 - 47
  • [2] The expression of CXCR4 is down-regulated on the CD34+cells of patients with myelofibrosis with myeloid metaplasia
    Rosti, Vittorio
    Massa, Margherita
    Vannucchi, Alessandro M.
    Bergamaschi, Gaetano
    Campanelli, Rita
    Pecci, Alessandro
    Viarengo, Glanluca
    Meli, Valentina
    Marchetti, Monia
    Guglielmelli, Paola
    Bruno, Edward
    Xu, Mingjiang
    Hoffman, Ronald
    Barosi, Giovanni
    BLOOD CELLS MOLECULES AND DISEASES, 2007, 38 (03) : 280 - 286
  • [3] Detection of aberrant signaling pathways in CD34+cells from patients with myelofibrosis with myeloid metaplasia using oligonulceotide microarrays.
    Jones, LC
    Tefferi, A
    Wachsman, W
    Hofmann, WK
    Koeffler, HP
    BLOOD, 2001, 98 (11) : 626A - 626A
  • [4] Reduced CD26 dipeptidylpeptidase IV membrane expression and activity of CD34+cells from patients with myelofibrosis with myeloid metaplasia.
    Campanelli, Rita
    Massa, Margherita
    Rosti, Vittorio
    Bergamaschi, Gaetano
    Viarengo, Gianluca
    Marchetti, Monia
    Hoffman, Ronald
    Barosi, Giovanni
    BLOOD, 2006, 108 (11) : 1033A - 1033A
  • [5] Does cytogenetic mosaicism in CD34+CD38low cells reflect the persistence of normal primitive hematopoietic progenitors in myeloid metaplasia with myelofibrosis?
    Bilhou-Nabéra, C
    Brigaudeau, C
    Clay, D
    Andrieux, J
    Lai, JL
    Brouty-Boyé, D
    Vignon, C
    Gharbi, MJ
    Le Bousse-Kerdilès, MC
    Praloran, V
    BLOOD, 2003, 102 (04) : 1551 - 1552
  • [6] Diagnostic and clinical relevance of the number of circulating CD34+ cells in myelofibrosis with myeloid metaplasia
    Barosi, G
    Viarengo, G
    Pecci, A
    Rosti, V
    Piaggio, G
    Marchetti, M
    Frassoni, F
    BLOOD, 2001, 98 (12) : 3249 - 3255
  • [7] Low number of circulating CD34+cells identifies a group of patients with myelofibrosis with myeloid metaplasia (MMM) who efficiently responds to STI571.
    Piaggio, G
    Castello, S
    Podesta, M
    Pozzi, S
    Pitto, A
    Rosti, V
    Rossi, E
    Bacigalupo, A
    Barosi, G
    Alberti, D
    Frassoni, F
    BLOOD, 2001, 98 (11) : 628A - 628A
  • [8] Clinical relevance of the number of circulating CD34-positive cells in myelofibrosis with myeloid metaplasia.
    Barosi, G
    Viarengo, G
    Rosti, V
    Piaggio, G
    Marchetti, M
    Frassoni, F
    BLOOD, 2000, 96 (11) : 744A - 745A
  • [9] Enhanced HDAC (histone deacetylase) enzyme activities in blood CD34+ cells in patients with myeloid metaplasia myelofibrosis (MMM).
    Wang, Jen-Chin
    Chen, Chi
    Dumlao, Theresa
    Chang, Thong
    Xiao, Yng-Li
    Sominsky, Inna
    Burton, Jack
    BLOOD, 2007, 110 (11) : 238B - 238B
  • [10] Circulating CD34+cells of primary myelofibrosis patients contribute to myeloid-dominant hematopoiesis and bone marrow fibrosis in immunodeficient mice
    Saito, Noriyuki
    Yamauchi, Takuji
    Kawano, Noriaki
    Ono, Rintaro
    Yoshida, Shuro
    Miyamoto, Toshihiro
    Kamimura, Tomohiko
    Shultz, Leonard D.
    Saito, Yoriko
    Takenaka, Katsuto
    Shimoda, Kazuya
    Harada, Mine
    Akashi, Koichi
    Ishikawa, Fumihiko
    INTERNATIONAL JOURNAL OF HEMATOLOGY, 2022, 115 (02) : 198 - 207
123