Pathophysiology of reduced glomerular filtration rate in delayed graft function

被引：0
|
作者：
Scandling, JD
Myers, BD
机构：
[1] STANFORD UNIV, MED CTR, DIV NEPHROL, SCH MED, DEPT MED, STANFORD, CA 94305 USA
[2] STANFORD UNIV, SCH MED, RENAL TRANSPLANT PROGRAM, STANFORD, CA 94305 USA
来源：
CURRENT OPINION IN NEPHROLOGY AND HYPERTENSION | 1997年 / 6卷 / 04期
关键词：
D O I：
暂无
中图分类号：
R5 [内科学]; R69 [泌尿科学（泌尿生殖系疾病）];
学科分类号：
1002 ; 100201 ;
摘要：
Delayed graft function is a form of postischemic acute renal failure. It lowers glomerular filtration rate in large part by depressing the glomerular transcapillary hydraulic pressure difference, the driving force for the formation of filtrate. Loss of proximal tubule cell polarity, impaired sodium reabsorption and tubuloglomerular feedback-mediated afferent arteriolar constriction are all implicated. A link between delayed graft function and chronic allograft injury is evident. The ensuing impairment of graft survival makes urgent the need for further elucidation of delayed graft function and a search for an effective therapy.
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页码：405 / 409
页数：5
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