A soluble form of Ldl receptor gene family member, Lr11, is essential for angiotensin II-induced Smc migration

被引：0

作者：

Pang, Meizi ^{[1
]}

Bujo, Hideaki ^{[1
]}

Ohwaki, Kenji ^{[1
]}

Takahashi, Mao ^{[2
]}

Shirai, Kohji ^{[2
]}

Schneider, Wolfgang J. ^{[3
]}

Saito, Yasushi ^{[1
]}

机构：

[1] Chiba Univ, Grad Sch Med, Chiba, Japan

[2] Toho Univ, Chiba 2748510, Japan

[3] Univ Vienna, BioCtr, Vienna, Austria

来源：

CIRCULATION | 2007年 / 116卷 / 16期

关键词：

D O I：

暂无

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

1449

引用

页码：298 / 298

页数：1

共 50 条

[1] Secreted soluble form of Lr11, an Ldl receptor gene family member, is a potent regulator of smooth muscle cell migration
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[2] Secreted soluble form of LR11, an LDL receptor gene family member, is a potent regulator of macrophage adhesion and smooth muscle cell migration
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[3] LR11, an LDL receptor gene family member, is a novel regulator of smooth muscle cell migration
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[4] Enhanced expression of the LDL receptor family member LR11 increases migration of smooth muscle cells in vitro
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[5] LR11, A NOVEL CIRCULATING MARKER OF CAROTID ATHEROSCLEROSIS, IS ESSENTIAL FOR ANGIOTENSIN II-INDUCED INTIMAL SMOOTH MUSCLE CELL MIGRATION
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[6] LR11 - A novel LDL receptor family member from man, rabbit and chicken
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[7] Expression of LR11, a mosaic LDL receptor family member, is markedly increased in atherosclerotic lesions
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[8] Lr11, an Ldl receptor family member, inhibits adipocyte maturation through the induced stabilization of beta-catenin
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[9] Circulating levels of soluble form of LR11, a novel migration regulator of intimal SMC, are increased in subjects with coronary stenosis
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[10] LR11, a mosaic LDL receptor family member, mediates the uptake of ApoE-rich lipoproteins in vitro
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