Three kinds of modulations of P/Q-type (Cav2.1) Ca2+ channel activity by metabotropic glutamate receptor subtype 1a (mGluR1a)

被引：0
|
作者：
Yamazaki, K
Wakamori, M
Shigetada, N
Mori, Y
机构：
[1] Kyoto Univ, Fac Engn, Dept Synthet & Biochem, Kyoto 6158510, Japan
[2] Osaka Biosci Inst, Osaka 5650874, Japan
来源：
JOURNAL OF PHARMACOLOGICAL SCIENCES | 2006年 / 100卷
关键词：
D O I：
暂无
中图分类号：
R9 [药学];
学科分类号：
1007 ;
摘要：
引用
收藏
页码：243P / 243P
页数：1
相关论文
共 50 条
  • [1] Multiple phases of modulations of P/Q-type(Cav2.1)Ca2+ channel activity by metabotropic glutamate receptor subtype 1a (mGluR1a)
    Yamazaki, Koji
    Akiyama, Satoshi
    Numaga, Takuro
    Nakanishi, Shigetada
    Mori, Yasuo
    Wakamori, Minoru
    JOURNAL OF PHARMACOLOGICAL SCIENCES, 2007, 103 : 174P - 174P
  • [2] Transmitter release deficits at the neuromuscular synapse of mice with mutations in the Cav2.1 (α1A) subunit of the P/Q-type Ca2+ channel
    Plomp, JJ
    Van Den Maagdenberg, AM
    Ferrari, MD
    Frants, RR
    Molenaar, PC
    MYASTHENIA GRAVIS AND RELATED DISORDERS: BIOMCHEMICAL BASIS FOR DISEASE OF THE NEUROMUSCULAR JUNCTION, 2003, 998 : 29 - 32
  • [3] Direct interaction and functional coupling between metabotropic glutamate receptor subtype 1 and voltage-sensitive Cav2.1 Ca2+ channel
    Kitano, J
    Nishida, M
    Itsukaichi, Y
    Minami, I
    Ogawa, M
    Hirano, T
    Mori, Y
    Nakanishi, S
    JOURNAL OF BIOLOGICAL CHEMISTRY, 2003, 278 (27) : 25101 - 25108
  • [4] Pain sensitivity in mice lacking the Cav2.1α1 subunit of P/Q-type Ca2+ channels
    Luvisetto, S.
    Marinelli, S.
    Panasiti, M. S.
    D'Amato, F. R.
    Fletcher, C. F.
    Pavone, F.
    Pietrobon, D.
    NEUROSCIENCE, 2006, 142 (03) : 823 - 832
  • [5] Cav2.1 mutations associated with different forms of ataxia change P/Q-type Ca2+ channel function
    Wappl, E
    Koschak, A
    Poteser, M
    Sinnegger, MJ
    Walter, D
    Kraus, RL
    Groschner, K
    Glossmann, H
    Grabner, M
    Striessnig, J
    BIOPHYSICAL JOURNAL, 2002, 82 (01) : 352A - 352A
  • [6] Binding of Gαo N terminus is responsible for the voltage-resistant inhibition of α1A(P/Q-type, Cav2.1) Ca2+ channels
    Kinoshita, M
    Nukada, T
    Asano, T
    Mori, Y
    Akaike, A
    Satoh, M
    Kaneko, S
    JOURNAL OF BIOLOGICAL CHEMISTRY, 2001, 276 (31) : 28731 - 28738
  • [7] Functional consequences of P/Q-type Ca2+ channel Cav2.1 missense mutations associated with episodic ataxia type 2 and progressive ataxia
    Wappl, E
    Koschak, A
    Poteser, M
    Sinnegger, MJ
    Walter, D
    Eberhart, A
    Groschner, K
    Glossmann, H
    Kraus, RL
    Grabner, M
    Striessnig, J
    JOURNAL OF BIOLOGICAL CHEMISTRY, 2002, 277 (09) : 6960 - 6966
  • [8] Demonstration of Binding of Neuronal Calcium Sensor-1 to the Cav2.1 P/Q-Type Calcium Channel
    Lian, Lu-Yun
    Pandalaneni, Sravan R.
    Todd, Paul A. C.
    Martin, Victoria M.
    Burgoyne, Robert D.
    Haynes, Lee P.
    BIOCHEMISTRY, 2014, 53 (38) : 6052 - 6062
  • [9] Binding of Gαo N-terminus is responsible for the voltage-resistant inhibition of α1A (Q-type, Cav2.1) Ca2+ channels
    Kinoshita, M
    YAKUGAKU ZASSHI-JOURNAL OF THE PHARMACEUTICAL SOCIETY OF JAPAN, 2002, 122 (08): : 565 - 572
  • [10] A new β subtype-specific interaction in α1A subunit controls P/Q-type Ca2+ channel activation
    Walker, D
    Bichet, D
    Geib, S
    Mori, E
    Cornet, V
    Snutch, TP
    Mori, Y
    De Waard, M
    JOURNAL OF BIOLOGICAL CHEMISTRY, 1999, 274 (18) : 12383 - 12390
12345