CagA protein phosphorylation in H-pylori infected gastric epithelial cells may be related with reduced FAK activation and increased Pyk2 activity

被引：0
|
作者：
Panayotopoulou, E. G. [1 ]
Papadakos, K. [1 ]
Michopoulos, S. [2 ]
Mantzaris, G. [3 ]
Papatheodoridis, G. [4 ]
Mentis, A. F. [1 ]
Sgouras, D. N. [1 ]
Archimandritis, A. [4 ]
机构：
[1] Inst Pasteur Hellen, Athens, Greece
[2] Alexandra Hosp, Gastroenterol Clin, Athens, Greece
[3] Evangelismos Med Ctr, Gastroenterol Clin, Athens, Greece
[4] Univ Athens, Sch Med, Dept Internal Med 2, GR-11527 Athens, Greece
来源：
HELICOBACTER | 2008年 / 13卷 / 05期
关键词：
D O I：
暂无
中图分类号：
R57 [消化系及腹部疾病];
学科分类号：
摘要：
引用
收藏
页码：411 / 411
页数：1
相关论文
共 15 条
  • [1] PYK2 interaction with CagA protein is concomitant with PYK2 increased tyrosine phosphorylation and reduced paxillin expression in H-pylori-infected gastric epithelial cells
    Panayotopoulou, E. G.
    Sgouras, D. N.
    Papadakos, K.
    Tzathas, C.
    Mentis, A.
    HELICOBACTER, 2010, 15 (04) : 319 - 320
  • [2] Helicobacter pylori CagA protein phosphorylation in early infection stages of gastric epithelial cells coincides with FAK dephosphorylation and is compensated by increased Pyk2 activity
    Panayotopoulou, E.
    Papadakos, K.
    Gonzalez, B. M.
    Michopoulos, S.
    Mantzaris, G.
    Archimandritis, A.
    Mentis, A.
    Sgouras, D.
    FEBS JOURNAL, 2008, 275 : 274 - 274
  • [3] Effect of caga tyrosine phosphorylation in MMPs production by H-pylori infected gastric epithelial cells
    Ryu, Eunju
    Lee, Yong Chan
    Lee, Inohk
    Nam, Young-Hee
    Lee, Seung-Taek
    GASTROENTEROLOGY, 2006, 130 (04) : A523 - A524
  • [4] H-pylori activates a Ca2+-dependent PYK2 in normal human gastric mucous epithelial cells
    Marlink, KM
    Bacon, KD
    Sheppard, BC
    Cover, TL
    Deveney, CW
    Rutten, MJ
    FASEB JOURNAL, 2002, 16 (04): : A78 - A78
  • [5] H.pylori induces phosphorylation of PYK2 that is associated with paxillin in normal human gastric mucous epithelial cells
    Bacon, K
    Marlink, K
    Deveney, K
    Sheppard, B
    Deveney, C
    Rutten, M
    GASTROENTEROLOGY, 2003, 124 (04) : A586 - A586
  • [6] H. PYLORI CAGA PROTEIN MAY BE INVOLVED IN THE INDUCTION OF EPITHELIAL TO MESENCHYMAL TRANSITION IN EXPERIMENTALLY IN VITRO INFECTED GASTRIC EPITHELIAL CELLS
    Sougleri, I. S.
    Papadakos, K. S.
    Mentis, A. F.
    Sgouras, D. N.
    HELICOBACTER, 2015, 20 : 76 - 76
  • [7] Cyclooxygenase-2 is upregulated in human gastric epithelial cells infected with H-pylori
    Kim, JM
    Jung, HC
    Kim, JS
    Ko, EJ
    Song, IS
    Kim, CY
    GASTROENTEROLOGY, 1999, 116 (04) : A215 - A215
  • [8] Role of protein ase-activated receptor-2 on cyclooxygenase-2 expression in H-pylori -: Infected gastric epithelial cells
    Seo, Ji Hye
    Kim, Kyung Hwan
    Kim, Hyeyoung
    SIGNAL TRANSDUCTION PATHWAYS, PT D: INFLAMMATORY SIGNALING PATHWAYS AND NEUROPATHOLOGY, 2007, 1096 : 29 - 36
  • [9] Helicobacter pylori CagA protein induces factors involved in the epithelial to mesenchymal transition (EMT) in infected gastric epithelial cells in an EPIYA- phosphorylation-dependent manner
    Sougleri, Ioanna S.
    Papadakos, Konstantinos S.
    Zadik, Mairi P.
    Mavri-Vavagianni, Mary
    Mentis, Andreas F.
    Sgouras, Dionyssios N.
    FEBS JOURNAL, 2016, 283 (02) : 206 - 220
  • [10] Apoptosis in gastric epithelial cells induced by H-pylori is accompanied by altered expression of the Bcl-2 protein family
    Chen, G
    Hansely, Z
    Ramey, W
    Holt, PR
    Moss, SF
    GUT, 1997, 41 : A16 - A16
12