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- [1] Notch1 signaling sensitizes tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis in human hepatocellular carcinoma cells by inhibiting Akt/Hdm2-mediated p53 degradation and up-regulating p53-dependent DR5 expression (Withdrawal of Vol 284, Pg 16183, 2009) JOURNAL OF BIOLOGICAL CHEMISTRY, 2021, 296
- [2] Erratum: Expression of Concern: Notch1 signaling sensitizes tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis in human hepatocellular carcinoma cells by inhibiting Akt/Hdm2-mediated p53 degradation and up-regulating p53-dependent DR5 expression (Journal of Biological Chemistry (2009) 284 (16183-16190) DOI: 10.1074/jbc.M109.002105) Journal of Biological Chemistry, 2020, 295 (26)
- [3] Notch1 Signaling Sensitizes Tumor Necrosis Factor-related Apoptosis-inducing Ligand-induced Apoptosis in Human Hepatocellular Carcinoma Cells by Inhibiting Akt/Hdm2-mediated p53 Degradation and Up-regulating p53-dependent DR5 Expression (Publication with Expression of Concern. See vol. 295, pg. 8881, 2020) (Withdrawn Publication. See vol. 296, 2021) (Withdrawn Publication. See vol. 296, 2021) (Withdrawn Publication. See vol. 296, 2021) JOURNAL OF BIOLOGICAL CHEMISTRY, 2009, 284 (24) : 16183 - 16190
- [4] Melittin, a major component of bee venom, sensitizes human hepatocellular carcinoma cells to tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced apoptosis by activating CaMKII-TAK1-JNK/p38 and inhibiting IκBα kinase-NFκB (Expression of Concern of Vol 284, Pg 3804, 2009) JOURNAL OF BIOLOGICAL CHEMISTRY, 2020, 295 (26) : 8880 - 8880
- [5] Melittin, a major component of bee venom, sensitizes human hepatocellular carcinoma cells to tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced apoptosis by activating CaMKII-TAK1-JNK/p38 and inhibiting IκBα kinase-NFκB (Withdrawal of Vol 284, Pg 3804, 2009) JOURNAL OF BIOLOGICAL CHEMISTRY, 2021, 296
- [6] The Synthetic Cannabinoid WIN 55,212-2 Sensitizes Hepatocellular Carcinoma Cells to Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand (TRAIL)-Induced Apoptosis by Activating p8/CCAAT/Enhancer Binding Protein Homologous Protein (CHOP)/Death Receptor 5 (DR5) Axis MOLECULAR PHARMACOLOGY, 2010, 77 (05) : 854 - 863
- [7] Role of Activating Transcription Factor 3 (ATF3) in Endoplasmic Reticulum (ER) Stress-induced Sensitization of p53-deficient Human Colon Cancer Cells to Tumor Necrosis Factor (TNF)-related Apoptosis-inducing Ligand (TRAIL)-mediated Apoptosis through Up-regulation of Death Receptor 5 (DR5) by Zerumbone and Celecoxib JOURNAL OF BIOLOGICAL CHEMISTRY, 2014, 289 (31) : 21544 - 21561
- [8] Melittin, a Major Component of Bee Venom, Sensitizes Human Hepatocellular Carcinoma Cells to Tumor Necrosis Factor-related Apoptosis-inducing Ligand (TRAIL)-induced Apoptosis by Activating CaMKII-TAK1-JNK/p38 and Inhibiting IκBα Kinase-NFκB (Publication with Expression of Concern. See vol. 295, pg. 8880, 2020) (Withdrawn Publication. See vol. 296, 2021) JOURNAL OF BIOLOGICAL CHEMISTRY, 2009, 284 (06) : 3804 - 3813