Genetic Deletion of Adenosine Triphosphate-Sensitive Potassium Channel Subunit Kir6.2 Paradoxically Confers Increased Tolerance to Myocyte Stress

被引：0

作者：

Henn, Matthew C. ^{[1
]}

Janjua, M. Burhan ^{[1
]}

Zhang, Haixia ^{[1
]}

Kanter, Evelyn M. ^{[1
]}

Makepeace, Carol M. ^{[1
]}

Schuessler, Richard ^{[1
]}

Nichols, Colin G. ^{[1
]}

Lawton, Jennifer S. ^{[1
]}

机构：

[1] Washington Univ, Sch Med, St Louis, MO USA

来源：

JOURNAL OF THE AMERICAN COLLEGE OF SURGEONS | 2015年 / 221卷 / 04期

关键词：

D O I：

10.1016/j.jamcollsurg.2015.07.043

中图分类号：

R61 [外科手术学];

学科分类号：

摘要：

引用

页码：S24 / S24

页数：1

共 37 条

[1] Severe congenital hyperinsulinism caused by a mutation in the Kir6.2 subunit of the adenosine triphosphate-sensitive potassium channel impairing trafficking and function
Marthinet, E
Bloc, A
Oka, Y
Tanizawa, Y
Wehrle-Haller, B
Bancila, V
Dubuis, JM
Philippe, J
Schwitzgebel, VM
JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 2005, 90 (09): : 5401 - 5406
[2] Increased tolerance to stress in cardiac expressed gain-of-function of adenosine triphosphate-sensitive potassium channel subunit Kir6.1
Henn, Matthew C.
Janjua, M. Burhan
Zhang, Haixia
Kanter, Evelyn M.
Makepeace, Carol M.
Schuessler, Richard B.
Nichols, Colin G.
Lawton, Jennifer S.
JOURNAL OF SURGICAL RESEARCH, 2016, 206 (02) : 460 - 465
[3] Diazoxide Cardioprotection Is Independent of Adenosine Triphosphate-Sensitive Potassium Channel Kir 6.1 Subunit in Response to Stress
Janjua, M. Burhan
Zhang, Haixia
Henn, Matthew C.
Makepeace, Carol M.
Kanter, Evelyn M.
Schuessler, Richard
Nichols, Colin C.
Lawton, Jennifer S.
JOURNAL OF THE AMERICAN COLLEGE OF SURGEONS, 2014, 219 (03) : S37 - S37
[4] Diazoxide Cardioprotection Is Independent of Adenosine Triphosphate-Sensitive Potassium Channel Kir6.1 Subunit in Response to Stress
Henn, Matthew C.
Janjua, M. Burhan
Zhang, Haixia
Kanter, Evelyn M.
Makepeace, Carol M.
Schuessler, Richard B.
Nichols, Colin G.
Lawton, Jennifer S.
JOURNAL OF THE AMERICAN COLLEGE OF SURGEONS, 2015, 221 (02) : 319 - 325
[5] Adenosine Triphosphate-Sensitive Potassium Channel Kir Subunits Implicated in Cardioprotection by Diazoxide
Henn, Matthew C.
Janjua, M. Burhan
Kanter, Evelyn M.
Makepeace, Carol M.
Schuessler, Richard B.
Nichols, Colin G.
Lawton, Jennifer S.
Journal of the American Heart Association, 2015, 4 (08): : e002016
[6] An Open Sarcolemmal Adenosine Triphosphate-Sensitive Potassium Channel Is Necessary for Detrimental Myocyte Swelling Secondary to Stress
Sellitto, Angela D.
Al-Dadah, Ashraf S.
Schuessler, Richard B.
Nichols, Colin G.
Lawton, Jennifer S.
CIRCULATION, 2011, 124 (11) : S70 - S74
[7] Myocyte volume and function in response to osmotic stress - Observations in the presence of an adenosine triphosphate-sensitive potassium channel opener
Mizutani, S
Prasad, SM
Sellitto, AD
Schuessler, RB
Damiano, RJ
Lawton, JS
CIRCULATION, 2005, 112 (09) : I219 - I223
[8] Adenosine triphosphate-sensitive potassium channel opener diazoxide maintains human myocyte volume homeostasis during stress
Sellitto, Angela D.
Maffit, Sara K.
Al-Dadah, Ashraf S.
Schuessler, Richard B.
Damiano, Ralph J.
Lawton, Jennifer S.
JOURNAL OF THE AMERICAN COLLEGE OF SURGEONS, 2009, 209 (03) : S29 - S29
[9] Myocytes with Gain-of-Function of Adenosine Triphosphate-Sensitive Potassium Channel Subunit Kir6.1 Have Altered Response to Stress of Hyperkalemic Cardioplegia
Henn, Matthew C.
Janjua, M. Burhan
Zhang, Haixia
Kanter, Evelyn M.
Makepeace, Carol M.
Schuessler, Richard
Nichols, Colin G.
Lawton, Jennifer S.
JOURNAL OF THE AMERICAN COLLEGE OF SURGEONS, 2015, 221 (04) : S25 - S25
[10] The E23K variant of KCNJ11 encoding the pancreatic β-cell adenosine 5′-triphosphate-sensitive potassium channel subunit Kir6.2 is associated with an increased risk of secondary failure to sulfonylurea in patients with type 2 diabetes
Sesti, Giorgio
Laratta, Emanuela
Cardellini, Marina
Andreozzi, Francesco
Del Guerra, Silvia
Irace, Concetta
Gnasso, Agostino
Grupillo, Maria
Lauro, Renato
Hribal, Marta Letizia
Perticone, Francesco
Marchetti, Piero
JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 2006, 91 (06): : 2334 - 2339

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