Effect of atmospheric particulate matter on the functional state of mitochondria

被引：1
|
作者：
V. Kondratyeva, Elena [1 ]
Vitkina, Tatyana I. [1 ]
机构：
[1] Res Inst Med Climatol & Rehabil Therapy, Far Eastern Sci Ctr Physiol & Pathol Resp, Vladivostok Branch, Lab Med Ecol & Recreat Resources, Vladivostok, Russia
来源：
RUSSIAN OPEN MEDICAL JOURNAL | 2023年 / 12卷 / 01期
关键词：
mitochondria; mitochondrial dysfunction; mitochondrial membrane potential; fatty acids; mitochondrial DNA damage; atmospheric particulate matter; DNA COPY NUMBER; OXIDATIVE STRESS; EXPOSURE; PM2.5; FINE; POLLUTION; DYSFUNCTION; APOPTOSIS; TOXICITY; DISEASE;
D O I：
10.15275/rusomj.2023.0106
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
The health risks associated with outdoor air pollution are of global concern. Atmospheric air pollution negatively affects a number of key aspects of human health, including the functioning of the respiratory, cardiovascular and central nervous systems, but many issues remain unresolved about the relationship between atmospheric air pollution and the development and course of pathologies. The review analyzes data from Russian and foreign sources on the effect of atmospheric particulate matter on the functional state of mitochondria. The effect of air pollution on structural changes in mitochondria, ATP synthesis, production of reactive oxygen species, damage to mitochondrial DNA, and mitochondrial membrane potential has been shown. The data presented in the review indicate the need for further studies of the functional state of mitochondria under the impact of solid particles in atmospheric air.
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页数：6
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